Titlebook: Microglia in Health and Disease; Marie-ève Tremblay,Amanda Sierra Book 2014 Springer Science+Business Media New York 2014 aging.microglia.

Atheroma

Priapism

URN

Developing and Mature Synapsestuning in a tightly regulated manner their morphology, gene expression, and functional behaviour. Depending on the nature of these changes, microglia can thicken and retract their processes, proliferate and migrate, release numerous signalling factors and compounds influencing neuronal physiology (e

SMART

nominal

congenial

Drug Addictionts. Historically, the biological basis of drug addiction has focused principally on neuronal activity. However, despite their pivotal role in the underlying pathology of drug addiction, neurons are not the only central nervous system (CNS) component involved. The role of additional cell types, espec

Prostaglandins

Agingr ramifications, beading and spheroid swellings, has been observed particularly in the cerebral cortex, as well as in and around the white matter. In aged animals, microglia express some surface antigens which are not normally present in their young counterparts, in addition to presenting altered mo

Intact

Neurodevelopmental and Neuropsychiatric Disordersneuropathology were recently reported to be closely associated with microglial dysfunction. Microglia, which are the major players of innate immunity in the central nervous system, respond rapidly to pathological changes, even minor ones, and contribute directly to neuroinflammation by producing var

insert

lacrimal-gland