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Titlebook: Microcirculation of the Heart; Theoretical and Clin Harald Tillmanns,Wolfgang Kübler,Horst Zebe Book 1982 Springer-Verlag Berlin Heidelberg

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發(fā)表于 2025-3-21 19:04:04 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Microcirculation of the Heart
副標(biāo)題Theoretical and Clin
編輯Harald Tillmanns,Wolfgang Kübler,Horst Zebe
視頻videohttp://file.papertrans.cn/634/633143/633143.mp4
圖書封面Titlebook: Microcirculation of the Heart; Theoretical and Clin Harald Tillmanns,Wolfgang Kübler,Horst Zebe Book 1982 Springer-Verlag Berlin Heidelberg
描述Microcirculation is a rather new field which has been of predominant interest to basic scientists, linking togeth~r technical, hemodynamic, and biochemical aspects. The fmdings elaborated, however, are not only of theoretical interest, but bear in addition great clinical implications. In clinical cardiology this became quite evident by the use of tracers in order to study myocardial perfuSion and by the deSCription of certain clinical entities - such as angina with normal coro- nary arteries - which are best explained by "disturbed microcirculation". With respect to this new developing theoretical and clinical field of cardiac microcirculation it was the aim of the Microcirculation Working Group of the European Society of Cardiology to have a symposium on which all different but clinically relevant aspects of cardiac microcirculation will be covered. This symposium, held in Heidelberg in January 1980, was planned not only for the exchange of concepts and ideas, but was expected to be in addition partially a teaching session; the basic scientists should be directed toward a better under- standing of the clinical problems, and the clinicians should learn more about the basic mechanis
出版日期Book 1982
關(guān)鍵詞Koronararterie; Koronarerkrankung; Mikrozirkulation; Mikrozirkulationsst?rung; angina pectoris; arteries;
版次1
doihttps://doi.org/10.1007/978-3-642-68485-2
isbn_softcover978-3-540-11346-1
isbn_ebook978-3-642-68485-2
copyrightSpringer-Verlag Berlin Heidelberg 1982
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 20:30:22 | 只看該作者
板凳
發(fā)表于 2025-3-22 03:45:07 | 只看該作者
Changes in Capillary Permeabilitythal. Nevertheless, during the course of these experiments, striking changes in capillary structure and function were demonstrated in severely ischemic left ventricular myocardium before and after reperfusion of arterial blood. These changes are the subject of this paper.
地板
發(fā)表于 2025-3-22 07:30:02 | 只看該作者
and biochemical aspects. The fmdings elaborated, however, are not only of theoretical interest, but bear in addition great clinical implications. In clinical cardiology this became quite evident by the use of tracers in order to study myocardial perfuSion and by the deSCription of certain clinical e
5#
發(fā)表于 2025-3-22 09:27:29 | 只看該作者
6#
發(fā)表于 2025-3-22 14:15:11 | 只看該作者
Rheological Aspectsascular diseases, it has now been shown that the terminal system of exchange vessels is not merely a passive irrigation system, but affects its own perfusion and the transmural exchange of metabolites by endogenous mechanisms that have largely evaded the quantification by the methods presently used in angiology and/or cardiology.
7#
發(fā)表于 2025-3-22 19:44:47 | 只看該作者
Flow Properties of the Bloodt is possible to neglect the fact that within the complex . the suspension properties of the blood as a whole may be relevant. On the contrary, these properties provide the basis for flow fractionation effects within the network which to a large extent determine the intranetwork distribution of erythrocyte flux and hence of oxygen delivery.
8#
發(fā)表于 2025-3-23 01:09:07 | 只看該作者
Coronary Microcirculationted perfused hearts treated with epinephrine [10]. Hence, if adenosine is a true mediator of metabolic regulation of CBF, the tissue concentration of the nucleoside should parallel the CBF when cardiac work (developed tension and/or heart rate) is changed. Studies were undertaken to investigate this relationship in different preparations.
9#
發(fā)表于 2025-3-23 03:06:08 | 只看該作者
Pathophysiologyction, causing the infarct to be larger or smaller. Unfortunately, as we will attempt to demonstrate, such a view is probably at variance with the actual three-dimensional pattern of a myocardial infarct and fails to take into account the nature of the microvasculature which determines whether an individual myocardial cell survives or dies.
10#
發(fā)表于 2025-3-23 06:10:58 | 只看該作者
Small Vessel DiseaseThe functional significance of “small vessel disease” of the heart receives very different treatment in clinical and pathologic investigations.
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