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Titlebook: Melanoma; Biologically Targete Ernest C. Borden (Director) Book 2002 Springer Science+Business Media New York 2002 Staging.angiogenesis.met

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發(fā)表于 2025-3-21 16:13:23 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Melanoma
副標(biāo)題Biologically Targete
編輯Ernest C. Borden (Director)
視頻videohttp://file.papertrans.cn/631/630292/630292.mp4
概述Includes supplementary material:
叢書名稱Current Clinical Oncology
圖書封面Titlebook: Melanoma; Biologically Targete Ernest C. Borden (Director) Book 2002 Springer Science+Business Media New York 2002 Staging.angiogenesis.met
描述Strategies of treatment involving therapeutic proteins, irnrnune immune cells, or cel- lular protein targets are those of greatest potential for further reducing mortality from melanoma. Therapeutic proteins or cells may inhibit melanoma cell growth either by augmentation of immune cell function or by inhibition of angiogenesis. Cytokines and melanoma antigens may be used either in vivo as a vaccine to stimulate irnrnune immune cell cell function or ex vivo to stimulate or proliferate cells for infusion. Alternatively, alteration in melanoma cell growth can occur through inhibition of protein signal transduction pathways within melanoma cells or in the endothelial cells constituting the necessary angiogenic support for tumor growth. The great promise of these therapies and their cellular targets constitutes the basis for Melanoma: Biologically Targeted Therapeutics. THE CLINlCAL PROBLEM More than four million people will be diagnosed with melanoma in the first decade of the 21st century. Half of those who will die will be individuals who would otherwise have had a life expectancy of another 25 years or more. These individuals will die of systemic systernic metastases, which are pre
出版日期Book 2002
關(guān)鍵詞Staging; angiogenesis; metastatic disease; surgery; tumor growth
版次1
doihttps://doi.org/10.1007/978-1-59259-159-6
isbn_softcover978-1-4684-9668-0
isbn_ebook978-1-59259-159-6Series ISSN 2364-1134 Series E-ISSN 2364-1142
issn_series 2364-1134
copyrightSpringer Science+Business Media New York 2002
The information of publication is updating

書目名稱Melanoma影響因子(影響力)




書目名稱Melanoma影響因子(影響力)學(xué)科排名




書目名稱Melanoma網(wǎng)絡(luò)公開度




書目名稱Melanoma網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Melanoma被引頻次




書目名稱Melanoma被引頻次學(xué)科排名




書目名稱Melanoma年度引用




書目名稱Melanoma年度引用學(xué)科排名




書目名稱Melanoma讀者反饋




書目名稱Melanoma讀者反饋學(xué)科排名




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A Pathologist’s Perspective on Prognostic Features of Malignant Melanomaw of pigment lesion pathology. It consists of benign nevus, including dysplastic nevus, . melanoma, and invasive melanoma. These are regarded as discrete entities with only minimal reference to a sequence of change from dysplasia to melanoma. Once invasive melanoma is diagnosed, the only additional
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Immunotherapy of Advanced Melanoma Directed at Specific Antigens with melanoma. Several lines of evidence may support this hypothesis: (.) in documented cases of children with spontaneous regression of cancer, melanoma has been found to be the cancer second in incidence to neuroblastoma .; (.) approximately 5% of patients with metastatic melanoma have an unknown
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Melanoma Antigensost simple approach seemed to be to inject subjects with crude tumor material, and there have been well over a hundred reports from 1902 through 1990 of patients being injected with tumor extracts . .Few of these reports are interpretable, however. Early studies in the 1950s using inbred strains of
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Interleukin-12uration factor .,.. It was identified as a factor secreted by Epstein Barr virus-transformed B cell lines. Its production can be induced by bacteria, intracellular parasites, viruses, or their products in T cell-dependent and -independent pathways. IL-12 has proinflammatory and immunoregulatory acti
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Interferonsntial role of biological strategies for melanoma. IFN-α2 is now licensed in more than 50 countries for more than a dozen clinical indications including melanoma. The clinical effectiveness of IFN-α2 has been established both in the adjuvant and metastatic clinical settings. IFNs were the first previ
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