Titlebook: Mechanisms of Secondary Brain Damage from Trauma and Ischemia; Recent Advances of o A. Baethmann,J. Eriskat,N. Plesnila Conference proceedi

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書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia影響因子(影響力)




書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia影響因子(影響力)學(xué)科排名




書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia網(wǎng)絡(luò)公開度




書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia被引頻次




書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia被引頻次學(xué)科排名




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書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia讀者反饋




書目名稱Mechanisms of Secondary Brain Damage from Trauma and Ischemia讀者反饋學(xué)科排名

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Lessons from epidemiologic studies in clinical trials of traumatic brain injuryTirilizad Trials which enrolled over 1,100 patients. A major observation from that trial was that pretreatment hypotension or hypoxia could be unbalanced even in a large two arm blinded study. Another issue of the tirilazad trial was the influence of gender affecting not only outcome but also drug metabolism.

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Pharmacological preconditioning in global cerebral ischemia function. Bcl-2-protein is expressed in neurons that survive cerebral ischemia and may parallel the time course of tolerance after ischemic preconditioning. With this study we examined whether differences in bcl-2-protein expression compared to baseline may be involved in the induction of ischemic tolerance using 3-NPA.

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