Titlebook: Mechanisms of Secondary Brain Damage; Current State Alexander Baethmann,Oliver Kempski,Ludwig Schürer Conference proceedings 1993 Springer-

Pericarditis

and , Control of Acid-Base Regulation of Brain Cells During Ischemic and Selective Acidic Exposureing only minimally from resting baseline levels over several minutes’ time. Similar but less pronounced effects follow direct electrical stimulation. The net change represents complex biophysical transmembrane and buffering mechanisms that appear to guard neuronal homeostasis..Studies carried out on

chlorosis

Mediators of Vascular and Parenchymal Mechanisms in Secondary Brain Damagerease of the AA concentration in the interstitial space. However, convincing therapeutic results on inhibition of AA are still lacking. In addition to the formation of vasogenic edema AA has been found to induce cytotoxic edema. From experiments dealing with the vasomotor effects Ellis .. (Am J Phys

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fulmination

Cerebral Protection by Adenosineinflux are controlled focusing in particular on the role of adenosine. This nucleoside is released from nerveand glial cells in larger amounts after ischemia. It counteracts at increased extracellular concentration the generation of burst discharges, an effect which is ascribed to a modulation of th

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Oration

Recovery of Brain Function Following Ischemiaacted recoveries, the potential for relearning in nervous circuits may play a part, but in more acute circumstances, for example in the progressive recovery from vasospasm, re-learning is clearly not a factor, and this demonstrates quite evidently that neurons at one moment apparently non-functionin

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ALB

rheumatology

Blood Flow and Clinical Course in Patients with Ischemic Stroke without Cerebrospecifíc Therapyion) despite pathologic neurologic findings was observed in 8/34 patients on day 3–7. Eight patients presented on day 3–7 with normal flow which later became ischemic again without evidence of another symptomatic episode. Correlation between severity of clinical findings and actual rCBF was low from

Carcinoma

Assessment of Emergency Care in Trauma Patientsation on the patient. The trauma evaluation score already used in the US became also a valid instrument in West-Germany. It shows a high correlation between survival and the patients’ physiological status in the field. Standard curves could be established for comparing individual or regional trauma