Titlebook: Mechanisms of Heart Failure; Pawan K. Singal,Ian M. C. Dixon,Naranjan S. Dhalla Book 1995 Springer Science+Business Media New York 1995 An

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Metabolic Changes in Nonischemic Myocardium During Pump Failurepletely resolved. The aim of this study was to understand metabolic changes in nonischemic myocardium on acute myocardial infarction in canine hearts in terms of the biochemistry of the sarcoplasmic reticulum, tissue ATP content, and the mitochondria.

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troponins

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Improved Preservation of Myocardial Ultrastructure in Perfusion-Fixed Human Heart Explants well-perfused areas with no tissue separation for subsequent electron microscopic evaluation. The control heart was indistinguishable from similar animal preparations. Details of nuclear pores, M lines, mitochondria, sarco-plasmic reticulurn, intercalated discs, intermediate filaments, and other fi

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Involvement of Leukotoxin in the Pathogenesis of Patients with Extensive Burnsn surface area under 70%. Maximum plasma leukotoxin concentrations significantly correlated with burn surface area. Significant correlation was also found between maximum plasma leukotoxin concentrations and those of C-reactive protein. Leukotoxin has been revealed to decrease blood pressure and lef

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Differential Regulation of Calmodulin-Dependent Cyclic Nucleotide Phosphodiesterase Isozymeshree other members — brain 60 kD CaMPDE, heart CaMPDE, and lung CaMPDE — show differential activation by calmodulin and Ca.. These observations are consistent with the notion that differential regulation by calmodulin and Ca. kinetics inhibition are important functions of these isozymes that provide

小木槌

Adriamycin Cardiomyopathy and Its Prevention in an Animal Modelcin) is a potent anthracyclin antitumor antibiotic, used for the treatment of a number of human malignancies. A life-threatening chronic side effect of Adriamycin treatment is the development of severe cardiomyopathy and ultimately congestive heart failure [1,2]. These changes are dose dependent [2]