Titlebook: Mechanisms of Heart Failure; Pawan K. Singal,Ian M. C. Dixon,Naranjan S. Dhalla Book 1995 Springer Science+Business Media New York 1995 An

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A. Martin Gerdes,Jan H. Prokosch-Graves,Harry E. Settles,James A. Burbach,Douglas D. Schocken gas of particles, a beam of photons, a heat bath ... The aim of quantum open system theory is to study the behaviour of this coupled system and in particular the dissipation of the small system in favour of the large one.978-3-540-30992-5978-3-540-33966-3Series ISSN 0075-8434 Series E-ISSN 1617-9692

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Kazuhiro Kosaka,Kohji Suzuki,Mika Hayakawa,Satoru Sugiyama,Takayuki Ozawa gas of particles, a beam of photons, a heat bath ... The aim of quantum open system theory is to study the behaviour of this coupled system and in particular the dissipation of the small system in favour of the large one.978-3-540-30992-5978-3-540-33966-3Series ISSN 0075-8434 Series E-ISSN 1617-9692

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Rajendra K. Sharma,Jawahar Kalra,Yingchun Tan gas of particles, a beam of photons, a heat bath ... The aim of quantum open system theory is to study the behaviour of this coupled system and in particular the dissipation of the small system in favour of the large one.978-3-540-30992-5978-3-540-33966-3Series ISSN 0075-8434 Series E-ISSN 1617-9692

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Tumor Necrosis Factor-Alpha in Chronic Heart Failuretory agents. TNF-α is a mediator of general inflammation and is believed to be the primary mediator of septic shock. It induces fever and promotes the infiltration of leukocytes in the site of infection and the expression of several adhesion molecules, such as integrins and intercellular adhesion mo

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Myocardial Ribonuclease Activity in Heart Failureog hearts (15.0 ±4.1 and 38.3 ± 6.4 units/esc), 14 failing compared with 13 nonfailing turkey hearts (30.1 ±4.7 and 39.3 ± 4.4units/sec), and 9 failing compared with 5 nonfailing chicken hearts (17.0 ± 3.5 and 25.7 ± 5.4units/sec) In all P < 0.0001). These data indicate that in HF produced from vari

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Vascular α-Adrenergic Function in Congestive Heart Failure to prolonged sympathetic stimulation, we have demonstrated that vascular α-adrenoceptors are desensitized and downregulated in a rat model of ischemic heart failure. Furthermore, we have also shown that in patients with severe congestive heart failure, both α.- and α.-adrenoceptor responsiveness is