Titlebook: Measurements in Musculoskeletal Radiology; Victor N. Cassar-Pullicino,A. Mark Davies Book 2020 Springer-Verlag GmbH Germany, part of Sprin

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Carlo Martinoli M.D.,Ali Attieh M.D.,Alberto Tagliafico M.D.al nonlinear programming methods. Fortunately, most of these problems exhibit a d.c. structure, by exploiting which it is possible in several cases of interest to devise reasonably practical algorithms for locating a global optimum to sufficient accuracy in acceptable computing time. A typical examp

Condescending

Filip M. Vanhoenacker M.D., Ph.D.,Koenraad L. Verstraete M.D., Ph.D.ic characteristics of the microbial ecotypes. The development of new powerful tools from molecular biology (such as polymerase chain reaction [PCR], simple and effective cloning and electrophoretic systems [SSCP, DGGE, TGGE] for nucleic acids separation, automated DNA sequencing facilities and wide

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e. In the UK and Greece, the numbers and distribution of heterotrophic bacteria were not strongly related to seasonal changes in temperature and rainfall. At Portchester Castle, qualitative changes in bacterial populations have been observed; actinomycetes were found only on decayed stone and domina

Dendritic-Cells

Clare Roche M.D,M.B,B.C.H,B.A.O,M.R.C.P.I and below the surface of monuments and objects of art. These biofilms can be very detrimental and thus quickly destroy the substrate. However, under certain conditions, the microflora contributes to the formation of a stabilised skin or layer. Slow growing poikilotrophic microorganisms are importan

GLUT

Naomi Winn,Eva Llopis,Victor N. Cassar-Pullicinomatter on artistic stoneworks can be attributed to inadequate past restorations, to the lysis of microbial cells of the primary surface colonisation, and to the presence of hydrocarbons originating from oil combustion. The latter appears to be a serious danger for the preservation of artworks themse

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Mario Padrón M.D.,Eugenie Sánchez M.D.,Victor N. Cassar-Pullicinonts in the development of this disease, substantial . evidence links the production of oxidized forms of LDL to molecular processes involved in atherogenesis (1–7). Depending on how LDL oxidation is initiated and the level of damage produced, a spectrum of damaged particles and biological effects ar

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Jean-Luc Drapé,Nicolas Theumannd world, claiming over 960,000 lives annually in the United States alone (1). A wealth of data now links serum cholesterol, in particular low-density lipoprotein (LDL) cholesterol (2) to the development of atherosclerosis. Despite this causative association between LDL cholesterol and atherosclerosi

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