Titlebook: Maturation Phenomenon in Cerebral Ischemia IV; Apoptosis and/or Nec Nicolas G. Bazan,Umeo Ito,Igor Klatzo Conference proceedings 2001 Sprin

Detrusor-Muscle

書目名稱Maturation Phenomenon in Cerebral Ischemia IV影響因子(影響力)




書目名稱Maturation Phenomenon in Cerebral Ischemia IV影響因子(影響力)學科排名




書目名稱Maturation Phenomenon in Cerebral Ischemia IV網(wǎng)絡(luò)公開度




書目名稱Maturation Phenomenon in Cerebral Ischemia IV網(wǎng)絡(luò)公開度學科排名




書目名稱Maturation Phenomenon in Cerebral Ischemia IV被引頻次




書目名稱Maturation Phenomenon in Cerebral Ischemia IV被引頻次學科排名




書目名稱Maturation Phenomenon in Cerebral Ischemia IV年度引用




書目名稱Maturation Phenomenon in Cerebral Ischemia IV年度引用學科排名




書目名稱Maturation Phenomenon in Cerebral Ischemia IV讀者反饋




書目名稱Maturation Phenomenon in Cerebral Ischemia IV讀者反饋學科排名

圖表證明

Bcl-w Expression and Localization in Brain Ischemianfarct regions. At the mechanistic level, incubation of isolated brain mitochondria with the addition of recombinant BAX or high concentrations of calcium resulted in release of cytochrome c from the mitochondria. In the presence of recombinant Bcl-w protein, however, the release of cytochrome c ind

合唱團

Repatriate

Interdict

Environmental Influence on Neuronal and Dendritic Spine Plasticity After Permanent Focal Brain Ischeand area borders of the cortex. Nonlesioned rats in the enriched environment had significantly more dendritic spines than rats in standard cages, in all layers studied. Individual spine morphology was significantly different, with more branched spines and an increase of spine head and neck diameter,

floodgate

Relationship Between DNA Fragmentation, Energy State, and Protein Synthesis After Transient Focal Cethose areas in which CPS did not return, covering 19±17% and 19±19% of contralateral hemisphere after 30 min MCAO, 27±9% and 32±9% after 60 min of MCAO, and 58±7% and 59±19% after 2 h MCAO. These data suggest that secondary energy failure after transient MCAO is heralded by persisting inhibition of

GORGE

無畏

semble

Upregulation of Transforming Growth Factor-Beta 1 Expression in Rat Hippocampal Neurons After Transil CA1 neurons can express TGF-β1 under physiological conditions and upregulate its expression during the first hours after ischemia, which is independent of glial activation. When neuronal damage increased, from 2 days to 4 days after ischemia, TGF-β1 immunoreactivity disappeared in damaged but rema

Hemiplegia