Titlebook: Management of Acute Pulmonary Embolism; Stavros V. Konstantinides (Professor of Medicine) Book 2007 Humana Press 2007 Atmen.embolism.hyper

CRUDE

Contemporary Diagnostic Algorithm for the Hemodynamically Stable Patient With Suspected Pulmonary Emgraphy, the historical gold standard in PE diagnosis. Therefore, modern diagnostic strategies for PE rely on combinations of noninvasive tests such as plasma D-dimer measurement, lower limb venous compression ultrasonography, ventilation-perfusion lung scan, and/or helical computed tomography, the r

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Contemporary Diagnostic Algorithm for the Hemodynamically Unstable Patient With Suspected Massive Puis. Management of the unstable, hypotensive patient with suspected massive PE poses a great challenge to the skills of the clinician in the intensive care unit or the emergency room, and it should direct the focus on rapid institution of treatment rather than absolute diagnostic certainty. Although

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Heparin Anticoagulation continuous intravenous infusion and activated partial thromboplastin time monitoring, or on low-molecular-weight heparin (LMWH) given by once- or twice-daily subcutaneous injections. There have been several studies comparing the use of UFH and LMWH for the treatment of deep vein thrombosis (DVT), a

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Thrombolysisy rate ranging between 1 and 2%. In contrast, more than 25% of patients with massive PE, defined by persistently low blood pressure or shock at presentation, die during the first 2 wk after diagnosis. Available evidence indicates that thrombolytic treatment reduces the mortality risk in these patien

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Foreknowledge

錯誤

Interventional Approaches to the Treatment of Acute Massive Pulmonary Embolismsystemic fibrinolysis because of major contraindications. Moreover, large-scale registries have shown that surgical embolectomy is carried out in only 1% of patients with massive PE and cardiogenic shock. Therefore, catheter-based thrombectomy or thrombus aspiration, as described in the present chap

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Initiative

Hereditary and Acquired Thrombophiliaa. Hereditary risk factors are detectable in approx 50% of patients with VTE events and a positive family history. Deficiencies of antithrombin, protein C, and its cofactor protein S, were the first disorders found to increase the risk of recurrent thrombotic events among carriers. Among Caucasians,

柏樹

Venous Thromboembolism and Cancernteractions between tumor cells and the hemostatic system. In addition, clinical risk factors for thromboembolism are frequently present in patients with cancer and include prolonged immobilization (especially during the hospital stay), surgery, and chemotherapy with or without adjuvant hormone ther