Titlebook: Liver Carcinogenesis; The Molecular Pathwa George G. Skouteris Conference proceedings 1994 Springer-Verlag Berlin Heidelberg 1994 Cancer Re

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constitutional

CANE

Stem Cell Activation in the Acetylaminofluorene-Treated Regenerating Rat Liver: A Bile Ductular Rea of normally proliferativekly quiescent hepatocytes into the cell cycle (Wright and Alison 1984; Alison 1986). Liver regeneration is commonly studied in the rat after a two-thirds partial hepatectomy. However, when hepatocyte regeneration is impaired, then relatively undifferentiated cells emerge fr

江湖騙子

瑣碎

APNEA

Preneoplastic Changes During Non-Genotoxic Hepatocarcinogenesis,pounds tested in preclinical carcinogenicity studies are usually non-genotoxic. The non-genotoxicity of a compound is operationally defined by l)its inability to cause mutations in several bacterial chromatid exchange and 2) its inability to produce covalent DNA adducts or induce DNA repair (measure

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控制

,Liver γ-Glutamyl Transpeptidase Activity after Cyclosporine A and Amlodipine Treatment,glutamyl compounds to a variety of amino acids and peptides. It has also been suggested that the action of GGT may be involved in protein synthesis, amino acid transport, collagen formation and degradation of peptides (Meister, 1974; Orlowski and Meister, 1970). The GGT, a heterogenous sialoglycopro

Immunotherapy

Enhanced Expression of the 27 Da Heat-Shock Protein During Dena-Induced Hepatocarcinogenesis in Ratporation into a series of specific polypeptides called “heat shock” or “stress” proteins (Hickey and Weber, 1982; Landry et al., 1982a; Welch, 1993) that seem to transiently protect the cell against further stress (Landry et al., 1982b).

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