Titlebook: Leukemia; Report of the Dahlem I. L. Weissman Conference proceedings 1985 Dr. S. Bernhard, Dahlem Konferenzen, Berlin 1985 cell.evolution.g

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Apoptosis

Phenotypic Heterogeneity and the Origins of Lymphoid Malignancy in Man: Biological and Clinical Impukemia phenotypes (excluding chromosome changes) appear to be minimally deviated replicas of normal ones. Uncoupling of proliferation from differentiation in acute leukemia results in the stabilization of progenitor cell phenotypes that would ordinarily be very transient. The phenotypic abnormality

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Multiple Mechanisms of Oncogene Activation in Viral and Nonviral Neoplasia,equences, gene amplification, translocation, and point mutations within regulatory or coding sequences. The mechanisms by which a specific proto-oncogene can most readily be activated probably reflects unique structural and biological characteristics of the gene involved. The c-. gene can be activat

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Tracing Development and Cell Lineages in Human Hemopoietic Neoplasia,tic neoplasms. The data indicate that chronic myelocytic leukemia (CML) is a clonal disease that involves a stem cell pluripotent for B lymphoid as well as myeloid cells. Although T cells may also arise from this stem cell, this question has not been definitively resolved. Elements that make up the