Titlebook: Lethal Arrhythmias Resulting from Myocardial Ischemia and Infarction; Proceedings of the S Michael R. Rosen (Professor of Pharmacology and

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On the Problem of Anisotropic Propagation in Ventricular Musclech are comprised of networks of membrane proteins fused together across the intercellular space at points of intimate cell-to-cell contact. Yet, even though gap junctional channels have a relatively low resistance [4–6], they represent a significant myoplasmic discontinuity [7–9], and propagation ma

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Anisotropic Reentry: A Model of Arrhythmias that may Necessitate a New Approach to Antiarrhythmic Dr excitation is one of these mechanisms [5, 6]. The role of reentry in the genesis of ischemic arrhythmias has been an area of intensive electrophysiologic investigation during the past 15 years, with a number of new and exciting experimental results being published. Among the most significant of the

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Role of Cytosolic Calcium in the Normal and Ischemic Heart: Potential New Insights from the Second Gardium to sequester or extrude calcium could have a variety of effects, the most serious of which would be the subversion of electrical activity in the nonischemic portion of the heart through the genesis of ventricular arrhythmias.

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