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Titlebook: Klinische Psychologie; Theoretische und Ide Johannes Helm,Hans-Dieter R?sler,Hans Szewczyk Book 1979 VEB Deutscher Verlag der Wissenschafte

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樓主: necrosis
21#
發(fā)表于 2025-3-25 04:57:31 | 只看該作者
Marlene Fuchs-Kittowskidouble-strand break repair. In this review, we summarize datafrom biochemical, cell based and in vivo approaches demonstrating that wild-type p53 restrains excessiveand mutagenic DNA exchange events. Thus, p53 appears to exhibit fidelity control of homologous recombination,and recent evidence also s
22#
發(fā)表于 2025-3-25 07:47:47 | 只看該作者
23#
發(fā)表于 2025-3-25 14:24:24 | 只看該作者
Klaus Groscheckption and replication. In 1998, it was found thathtopoI might play another important role in the cellular response to DNA damage -- the so-called htopoIdamage response. Since this initial discovery, many studies have suggested that the htopoI damage responseis involved in DNA repair as well as in ap
24#
發(fā)表于 2025-3-25 17:40:16 | 只看該作者
ption and replication. In 1998, it was found thathtopoI might play another important role in the cellular response to DNA damage -- the so-called htopoIdamage response. Since this initial discovery, many studies have suggested that the htopoI damage responseis involved in DNA repair as well as in ap
25#
發(fā)表于 2025-3-25 21:05:53 | 只看該作者
Johannes Helmctivity, it has been shown that it can undergostoichiometric (non-enzymatic) interactions with other proteins. This interaction is not only limited tothe tetrameric holoenzyme; the individual subunits of CK2, i.e. the catalytic α-subunit and the regulatoryβ-subunit, can also interact by themselves w
26#
發(fā)表于 2025-3-26 01:13:35 | 只看該作者
27#
發(fā)表于 2025-3-26 05:04:36 | 只看該作者
28#
發(fā)表于 2025-3-26 11:23:42 | 只看該作者
29#
發(fā)表于 2025-3-26 15:59:55 | 只看該作者
30#
發(fā)表于 2025-3-26 19:58:24 | 只看該作者
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