Titlebook: Kindling 5; Michael E. Corcoran,Solomon L. Moshé Book 1998 Plenum Press, New York 1998 Epilepsie.death.memory.neurochemistry.receptor

senile-dementia

Activities of Protein Kinase C in the Kindling Model of Epilepsyhroughout the life time of the animal. Kindling is an excellent model for studying the cellular mechanisms involved in the long-lasting neuronal excitability changes that underlie epilepsy, but the basic mechanism underlying this phenomenon still remains to be studied.

定點(diǎn)

SPURN

Long-Lasting Changes in the Pharmacology and Electrophysiology of Amino Acid Receptors in Amygdala Kntains moderate amounts of γ-aminobutyric acid (GABA). and GABA immunoreactivity.. Morphologically, interneurons comprising ~ 5% of the cells. have been classified as GABAergic. and form synapses with the principal cell types. Lesioning the afferent ST pathway did not reduce GABA content, suggesting GABA neurons in the BLA are intrinsic..

Stable-Angina

可行

滔滔不絕地講

Comparison of Synapse Remodeling Following Hippocampal Kindling and Long-Term Potentiations has been attained, cessation of stimulation for many months and even years does not result in the loss of the newly acquired abnormal reaction: reintroduction of the original stimulus reliably evokes a generalized seizure..Thus, kindling involves a virtually permanent augmentation of synaptic responsiveness in the stimulated circuit.

搏斗

subacute

Genetic Predisposition and Kindling Susceptibility in Primatesn when medication is withdrawn, while others begin with generalized convulsive seizures which readily recur when medication is reduced. Between these two extremes, there are those patients who display partial motor seizure without ever developing secondary generalization. The reason for such differe

Hallowed

Idiosyncrasies of Limbic Kindling in Developing Ratst kindling in a number of characteristics which may shed light on age-dependent differences in human epilepsy.,(Baram this volume). At all ages (older than postnatal day [PN] 7), repeated electrical stimuli delivered to limbic structures lead to the progression from focal to generalized seizures, in

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