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Titlebook: Ischemia-reperfusion in cardiac surgery; Hans Michael Piper,Claus J. Preusse Book 1993 Kluwer Academic Publishers 1993 Bypass.cardiac func

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發(fā)表于 2025-3-21 16:15:44 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Ischemia-reperfusion in cardiac surgery
編輯Hans Michael Piper,Claus J. Preusse
視頻videohttp://file.papertrans.cn/476/475532/475532.mp4
叢書名稱Developments in Cardiovascular Medicine
圖書封面Titlebook: Ischemia-reperfusion in cardiac surgery;  Hans Michael Piper,Claus J. Preusse Book 1993 Kluwer Academic Publishers 1993 Bypass.cardiac func
描述In open heart surgery, protection of the myocardium againstinjury by ischemia-reperfusion is an issue of utmost clinicalinterest. The knowledge on the causal mechanisms of myocardial injuryin ischemia-reperfusion has greatly progressed in recent years, bothto clinical and experimental research. This book provides a criticaland comprehensive overview on the current discussion of myocardialprotection in cardiac experimental research and surgery. Leadinggroups from Europe, the U.S. and Japan have contributed reviews oftheir special fields..On the basic science side, major topics discussed are the mechanismsof stunning and lethal reperfusion injury, the role of the endotheliumand leukocyte activation in postischemic myocardial dysfunction, andthe differences between the adult and neonatal myocardium. On theclinical side, the major concepts and procedures for cardioplegicmyocardial protection, their rationales, application and respectivemerits are discussed. In a third part of the book, possible approachesfor the monitoring of myocardial injury during ischemia andreperfusion are evaluated, with particular attention to theirintraoperative use..This book brings together the scientific dis
出版日期Book 1993
關(guān)鍵詞Bypass; cardiac function; cardiac surgery; cardiology; heart; physiology; thoracic surgery
版次1
doihttps://doi.org/10.1007/978-94-011-1713-5
isbn_softcover978-94-010-4750-0
isbn_ebook978-94-011-1713-5Series ISSN 0166-9842
issn_series 0166-9842
copyrightKluwer Academic Publishers 1993
The information of publication is updating

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沙發(fā)
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Moderate ischemic injury and myocardial stunningyocardial dysfunction or “myocardial stunning” [.–.]. At the other end are severe, irreversible abnormalities such as cell death (infarction). Thus, myocardial stunning should be considered as a mild, sublethal sequela of ischemia/reperfusion injury.
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Myocardial tissue pH in the assessment of the extent of myocardial ischemia and the adequacy of myocod of aortic clamping. Measurement of myocardial temperature can only reflect the adequacy of the delivery of the cardioplegia solution during the initial period following aortic clamping. Furthermore, temperature is no indication of the metabolic viability of the myocardium since one can cool even a dead heart!
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發(fā)表于 2025-3-22 16:57:03 | 只看該作者
Role of leukocytes in reperfusion injuryrfusion. Thus reperfusion injury refers to damage caused by the reinstitution of blood flow, in contrast to cell death caused by the preceding ischemic episode. This cellular damage may be reflected as structural, functional or metabolic derangements.
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