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Titlebook: Iron Physiology and Pathophysiology in Humans; Gregory J. Anderson,Gordon D. McLaren Book 2012 Springer Science+Business Media, LLC 2012 A

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發(fā)表于 2025-3-21 17:04:31 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Iron Physiology and Pathophysiology in Humans
編輯Gregory J. Anderson,Gordon D. McLaren
視頻videohttp://file.papertrans.cn/476/475402/475402.mp4
概述Excellent resource.Chapters are written by world renowned experts.Most current and up-to-date text
叢書名稱Nutrition and Health
圖書封面Titlebook: Iron Physiology and Pathophysiology in Humans;  Gregory J. Anderson,Gordon D. McLaren Book 2012 Springer Science+Business Media, LLC 2012 A
描述.Iron Physiology and Pathophysiology in Humans. provides health professionals in many areas of research and practice with the most up-to-date and well-referenced volume on the importance of iron as a nutrient and its role in health and disease.? This important new volume is the benchmark in the complex area of interrelationships between the essentiality of iron, its functions throughout the body, including its critical role in erythropoiesis, the biochemistry and clinical relevance of iron-containing enzymes and other molecules involved in iron absorption, transport and metabolism,? he importance of optimal iron status on immune function, and links between iron and the liver, heart, ?brain and other organs.? Moreover, the interactions between genetic and environmental factors and the numerous co-morbidities seen with both iron deficiency and iron overload in at risk populations are clearly delineated so that students as well as practitioners can better understand the complexities of these interactions. .Key features of the volume include an in-depth index and recommendations and practice guidelines are included in relevant chapters. The volume contains more than 100 detailed tables
出版日期Book 2012
關(guān)鍵詞Anemias; Cellular Metabolism; Cellular Metabolism; Homeostasis; Iron; Iron; Iron Homeostasis; Iron Overload
版次1
doihttps://doi.org/10.1007/978-1-60327-485-2
isbn_softcover978-1-4939-5765-1
isbn_ebook978-1-60327-485-2Series ISSN 2628-197X Series E-ISSN 2628-1961
issn_series 2628-197X
copyrightSpringer Science+Business Media, LLC 2012
The information of publication is updating

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bs) with the high potency of cytotoxic agents. In the first generation of ADCs, the toxic payload is attached to the mAb via chemical conjugation to endogenous lysine or cysteine residues providing only limited control over site specificity and drug-to-antibody ratio (DAR). The resulting product is
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hey may be of vegetable, animal or microbial origin, but in all cases specific toxicants can be generated by interaction between endogenous enzymes and substrates if processing releases cellularly bound materials. Substrates are commonly non-toxic in themselves but release toxicants as a result of a
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Weng-In Leong R.D., Ph.D.,Bo L?nnerdal Ph.D.evelopment of glucose-isomerising enzymes in an immobilised form has enabled the starch producers to convert economically 40–45 % of the dextrose in their high-dextrose syrups to fructose, thus producing a syrup which can match the sweetness of sucrose..This presentation concentrates on the other im
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