Titlebook: Intracranial Pressure II; Proceedings of the S N. Lundberg,U. Pontén,M. Brock Conference proceedings 1975 Springer-Verlag Berlin Heidelberg

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Co-chairman’s Summaryacranial pressure. To paraphrase him, on the surface these issues appear to be well understood, but to date the intimate details remain if not obscure at least somewhat hazy. These areas concern, . those factors which regulate the secretion of the cerebrospinal fluid, ., the relationship of the cere

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Formation of Cerebrospinal Fluidsystem for up to seven hours. The experimental system has been presented elsewhere in great detail (1) and will only be briefly described at this time. Arterial inflow to the plexus was controlled by means of a catheter in the internal carotid artery with ligation of all branches except the anterior

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Evidence for Bulk Flow of Cerebral Interstitial Fluid and its Possible Contribution to Cerebrospinal evidence (1) supporting the classical view of cerebrospinal fluid formation by brain, i.e. of a slow “l(fā)ymphatic” flow of ISF from brain to CSF via a perivascular canalicular system (2). This paper summarizes evidence suggesting bulk flow of ISF along the course of cerebral blood vessels and also pr

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Porous Nature of the Absorptive Mechanismng a variety of substances into the ventricular or subarachnoid space and estimating the rate of loss. This latter may be determined by examining the change in concentration of the CSF after a known time or by measuring the change in concentration in the blood; the blood sampled may be that from, sa

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CSF Hydrodynamics Studied by Means of Constant Pressure Infusion Techniquehe lumbar route. The resulting change in CSF pressure at one or two infusion rates has been recorded (1, 2,3,4,5,6). The constant rate infusion test according to KATZMAN and HUSSEY (2) was the starting point for the author who has performed 30 investigations with that method. There is one important

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Arachnoid Villi and Granulationsd him that these passages did not exist (1,2). The concept that the arachnoid villus is a blind diverticulum of arachnoid that projects into a venous channel, and that it serves as a membrane through which cerebrospinal fluid passes into the blood stream was then widely accepted, WELCH and FRIEDMAN

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The Definition of a Reduced CSF Absorption Syndrome: Clinical and Experimental Studies of the increase in intracranial pressure, and even which intracranial compartment is involved has, however, remained obscure. In a recent review of the available evidence it was argued that the rise in intracranial pressure is secondary to an increase in CSF volume as a result of impaired CSF absor

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CSF Dynamics: A Mathematical Approachseveral factors (elasticity of the vascular bed, CSF flow, sagittal sinus venous pressure (SSVP), vasomotor tone), which act together and cannot be isolated. Since 1969 (1,2) we have tried to study the above factors in a synthetic way and to elaborate a mathematical model for CSF dynamics.

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A Computer Model of CSF Dynamics formula holds true: ., where P. = pressure in the choroidal vessels, .. = oncotic pressure of the blood, P. = CSF pressure, and K. = a factor representing metabolic influences. The CSF pressure/absorption relationship was examined experimentally. The resulting curve for CSF pressure/ absorption rel

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Chairman’s Introduction intracranial compartments (brain tissue, intravascular blood, and CSF) at the time measurements are made to assess the volume-pressure relationship. For example, edema may expand the brain tissue compartment without increasing intracranial pressure (ICP) if the volume of CSF expressed from the intr