Titlebook: Intracranial Angiomas. Neurosurgical Intensive Care. Supratentorial Tumors in Children; Proceedings of the 4 Wolfgang Joachim Bock,Christia

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STELL

Embolization Techniques in the Treatment of Cerebral Arteriovenous Malformationsll diameter of the vessels than by the sharp angle of the bifurcations [1]. Further reductions in size (e.g. Tracker 10, Target Therapeutic Corporation, California, USA) nowadays allow even the most delicate vessels to be approached effectively, e.g., cerebral vessels in pediatric patients (Fig. 1) or perforating arteries.

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Results of Combined Endovascular and Surgical Treatment of Intracranial Arteriovenous Malformations vulnerable areas of the brain which are hardly or not at all accessible to surgical treatment. The development of endovascular superselective techniques has given us a powerful alternative [1, 5, 7]. The aim of treatment is radical occlusion of the AVM for prevention of bleeding.

dry-eye

Indications, Technique, and Results of Microsurgical Treatment of Intracranial Arteriovenous Malformhe vast majority of cases be completely excised in one session [2, 9, 10, 11, 14, 15, 16], which markedly reduces the risk of recurrent hemorrhage. The results of microsurgical excision have steadily improved [7], as we can demonstrate in our follow-up study of 57 patients with a brain AVM.

headlong

Surgical Problems in Partially Embolized Angiomasthe malformation. One further reason could be the lack of information on how effectively embolized but still active AVMs can be surgically treated. This article is intended to contribute to some aspects of the problems encountered during surgery on embolized AVMs and to the final result after surgical removal of an incompletely embolized AVM.

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Mechanisms of K+-induced Glial Swellingfrom high K.. results from intracellular accumulation of this ion secondary to activation of Na./K.-ATPase. Cell swelling under these conditions is supported by enhanced intracellular formation of lactic acid resulting from K..-induced stimulation of anaerobic metabolism.

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https://doi.org/10.1007/978-3-642-76182-9Gef??mi?bildung; Gehirntumor; Hirngef??; Hirngeschwulst; Hirngesch?digtes Kind; Intensivbehandlung; Neuroc

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ATP861

978-3-540-53311-5Springer-Verlag Berlin Heidelberg 1991