Titlebook: Intracellular Protein Catabolism II; Vito Turk,Neville Marks,J. Frederick Woessner Book 1977 J. Stefan Institute, Ljubljana, Yugoslavia 19

繁榮地區(qū)

有斑點(diǎn)

Irascible

Inhibition of Cell Protein Degradation by Microtubular Inhibitors,ers(2),(9),(11),(27), using other experimental systems, have corroborated these requirements for energy and protein synthesis in degradation of cell proteins, other studies(1),(8),(20),(26) have suggested that the requirement for protein synthesis appears to be less than absolute.

煤渣

Specificity of Breakdown Based on the Inactivation of Active Proteins and Peptides by Brain Proteolintracellular catabolism; thus a study of the specificity of the enzymes involved could help reveal some of the pathways involved. It is the purpose of this short account to focus attention on particular brain enzymes that are directly involved in this function with emphasis placed on contributions from the authors’ laboratory.

不持續(xù)就爆

Determination of the Average Degradation Rate of Mixtures of Protein,refore, varies with the duration of the experiment, becoming slower as the period of measurement becomes longer (1–4). We have attempted to obtain a unique value for the average rate of turnover of rat liver protein from the decay of label in protein after .CO. injection.

惡意

Nonporous

Protein Degradation in Neurospora Crassa, the rate of protein degradation increases by decreasing the growth rate and it is pronounced only in ethanol growing cells..During a shift-down transition of growth protein degradation is enhanced. 2-deoxyglucose prevents protein degradation in glucose starved cells.

perpetual

Studies on the Mechanism of Endocytosis in Fibroblasts with the use of Specific Antibodies,74). The succession of events involves an invagination of the plasma membrane, the formation of a closed vesicle, called a phagosome, and the subsequent fusion of this vesicle with lysosornes, leading to the formation of a digestive vacuole (Jacques, 1969).

Contort

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