Titlebook: Intensive Care Medicine; Annual Update 2003 Jean-Louis Vincent (Head) Conference proceedings 2003 Springer-Verlag Berlin Heidelberg 2003 Ac

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Human Genetics and Human Sepsis: Is the Tail Wagging the Dogisease is thought to be about one in ten. Of the estimated 500-million falciparum malaria infections per year, only about 20% are symptomatic and the overall mortality is probably only 1–2%. Approximately 10% of people carry . in their nasopharynx, yet only 1 in 40 000 develop invasive meningococcal

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The Effect of Alcohol Consumption on Risk for Sepsis and ARDS acute respiratory distress syndrome (ARDS). Chronic alcohol abuse similarly increases the mortality from ARDS. One possible mechanism by which chronic alcohol abuse may increase susceptibility to acute lung injury (ALI) is through alterations in pulmonary glutathione homeostasis. This chapter discu

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Reconciling Clinical Criteria and the Use of Genetically Engineered Animals in Sepsis Researchisition and evaluation of objective criteria and physiological parameters, respectively. The transformation of a typical clinical setting in intensive care units (ICUs), including mechanical ventilation and invasive monitoring of organ function into experiments on mice, may become routine maneuvers.

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Microarray Technology in Sepsis: Tool or Toy?tiple types of research studies comprising a broad panel of clinical aspects. One of the lessons learned to date is that evaluation of new sepsis therapies has been hampered by fairly unspecific, clinically-based inclusion criteria which insufficiently reflect the molecular mechanisms [1].

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Supplementing Arginine during Sepsis: from Theory to Clinical Practicery sources and by endogenous synthesis. In humans, arginine is considered a conditionally essential amino acid since stress conditions with increased arginine demand such as sepsis and inflammation indicate that exogenous arginine is required for a positive nitrogen balance.

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Role of Epithelial ICAM-1 in Endotoxin-Induced Lung Injuryepithelial cells express and secrete various immune molecules such as adhesion molecules, cytokines and chemokines. Through the expression and production of these inflammatory mediators, the airway epithelium is thought to play an important role in the initiation and exacerbation of inflammatory response within the airway.

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