Titlebook: Intensive Care Medicine; Annual Update 2002 Jean-Louis Vincent (Head) Book 2002 Springer Science+Business Media New York 2002 Trauma.anesth

PAC

弓箭

right-atrium

雕鏤

Molecular Mechanisms of Complement Activation during Ischemia and Reperfusionbeneficial effects of reperfusion of ischemic tissues in animal models. In this chapter we will summarize the role of inflammation in the pathogenesis of I/R-injury, and discuss in more detail that of the complement system, as this plasma cascade system seems to be a key mediator in this condition.

惡意

enlist

Pulmonary Endothelial Angiotensin Converting Enzyme Activity in Lung Injuryn of the lungs, and the tremendous surface area of the pulmonary capillary endothelium allows the latter to filter the entire circulating blood volume before it enters the systemic circulation. Thus, pulmonary endothelial functional and structural integrity are essential for adequate pulmonary and systemic cardiovascular homeostasis.

錫箔紙

High Volume Hemofiltration in Sepsisprovements. The question of treatment dose has appropriately been raised as it remains undefined and a matter of controversy. A large-scale clinical trial has clarified issues on treatment dose in acute renal failure but a sufficiently powered study on hemofiltration dose in sepsis is still lacking.

Musket

動物

溺愛

Cell Adhesion Molecules and Leukocyte Trafficking in Sepsis circulation to such foci results from interactions between leukocytes and endothelial cells, controlled by the coordinated expression of cell adhesion molecules (CAM) on both cell types. CAM mediate attachment of cells both to one another and to extracellular matrix. They include the selectins, int