Titlebook: Integration in Respiratory Control; From Genes to System Marc J. Poulin,Richard J. A. Wilson Book 2008 The Editor(s) (if applicable) and Th

asthma

Control of the Exercise Hyperpnea: The Unanswered Question.” I have chosen to sub-title my presentation, “The Unanswered Question” (with apologies to Charles Ives) in the hope that even if the answer does not eventuate then at least the nature of the essential question will. But space constraints allow only one theme to be developed and that only for moder

殘廢的火焰

A Peripheral Oxygen Sensor Provides Direct Activation of an Identified Respiratory CPG Neuron in ,in largely unknown. In order to study this topic at a fundamental level, we have developed a simple invertebrate model system, . wherein we have identified peripheral chemoreceptor cells (PCRCs) that relay hypoxiasensitive chemosensory information to a known respiratory CPG neuron, right pedal dorsa

Aboveboard

Environmental Hyperoxia and Development of Carotid Chemoafferent Functions nerve (CSN) and phrenic nerve responses to acute hypoxia. We determined the maximal CSN responses of anesthetized adult rats to severe hypoxia (ventilation with 100% N.) or asphyxia (stopped ventilator) after 1, 2, and 4 weeks of postnatal hyperoxia (60% O.) (PNH). As with acute responses to hypox

類人猿

Conflagration

Effect of Systemic Administration of the Nitric Oxide Synthase Inhibitor L-NMMA on the Human Ventilammune responses, cell adhesion and neurotransmission), its role in the regulation of respiration is less clear. In carotid body studies, most studies indicate that NO appears to play an inhibitory role in response to hypoxia (Gozal, Gozal, Gozal and Torres 1996a; Gozal, Torres, Gozal and Littwin 199

HARD

Effects of Volatile Anesthetics on Carotid Body Response to Hypoxia in Animalsic plus manual) for full-paper articles in English that used methodologies enabling any anesthetic effect to be located to the carotid body. We found just 7 articles that met our inclusion criteria, incorporating 16 separate studies. Anesthetic (mean dose ± SD 0.70 ± 0.33 MAC) significantly depresse

AGONY

Mutation of the von Hippel-Lindau Gene Alters Human Cardiopulmonary Physiologynvestigated the potential role of the VHL-HIF pathway in human systems-level physiology. Patients diagnosed with Chuvash polycythaemia, a rare disorder in which VHL signalling is specifically impaired, were studied during acute hypoxia and hypercapnia. Subjects breathed through a mouthpiece and vent

Keratectomy

Intravenous Endothelin-1 and Ventilatory Sensitivity to Hypoxia in Humansd twice to 4 hr eucapnic hypoxia. They received a continuous infusion of ET-1 during the ET-1 protocol and an infusion of saline during the control protocol. Plasma ET-1 levels and an index of ventilation were measured regularly. Hypoxia caused a rise in plasma ET-1 in the control protocol. Hypoxia

Intruder

Key Roles for AMP-activated Protein Kinase in the Function of the Carotid Body?excites the oxygen-sensing carotid body type 1 cells has not been fully defined. We have previously proposed that the enzyme adenosine monophosphateactivated protein kinase (AMPK) may couple hypoxic inhibition of mitochondrial oxidative phosphorylation to carotid body type I cell excitation (Evans,

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