Titlebook: Inflammation and Stroke; Giora Z. Feuerstein Book 2001 Springer Basel AG 2001 Inflammation research.ancillary brain diseases.brain.cogniti

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季雨

Inflammation as a risk factor for stroke: evidence from experimental models stroke in Sweden, stroke incidence exhibited seasonal variability with higher incidence in the colder winter months [1]. Infection was suggested to underlie the increased stroke incidence during winter since, in young adults without established stroke-risk factors, evidence of infection was seen co

arrhythmic

Salutary effect of autoimmune T cells after central nervous system injuryvolving restriction of local immune responses within the CNS. This phenomenon is probably an evolutionary adaptation developed to protect the intricate neuronal networks of the CNS from potentially disruptive incursion by the immune system [1–3]. An early definition of immune privilege was based on

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cravat

Cyclic activation and inactivation of brain vessels involving inflammatory mediators — implications cular tubes. Recent studies of endothelial cells in culture systems have proven these simplistic notions untenable and have shed light on the manifold functions of the endothelium under normal as well as pathological conditions. Endothelium can be characterized as a dynamic, heterogenous, disseminat

FIS

Do leukocytes play a role in focal ischemia in the brain? An objective review of the literaturece of neutrophils within ischemic tissue at the approximate time that substantial cell death occurs, (2) the reduction of ischemia-induced cell loss following neutropenia, and (3) the observation that treatments which prevent neutrophil trafficking can be neuroprotective. Despite the numerous studie

jocular

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