Titlebook: Inflammation and Lung Cancer; Steven M. Dubinett Book 2015 Springer Science+Business Media New York 2015 Inflammation.Lung cancer.Micro-RN

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書目名稱Inflammation and Lung Cancer影響因子(影響力)




書目名稱Inflammation and Lung Cancer影響因子(影響力)學(xué)科排名




書目名稱Inflammation and Lung Cancer網(wǎng)絡(luò)公開度




書目名稱Inflammation and Lung Cancer網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Inflammation and Lung Cancer被引頻次




書目名稱Inflammation and Lung Cancer被引頻次學(xué)科排名




書目名稱Inflammation and Lung Cancer年度引用




書目名稱Inflammation and Lung Cancer年度引用學(xué)科排名




書目名稱Inflammation and Lung Cancer讀者反饋




書目名稱Inflammation and Lung Cancer讀者反饋學(xué)科排名

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招募

978-1-4939-4511-5Springer Science+Business Media New York 2015

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抱怨

Sherven Sharma PhD,Siwen Hu-Lieskovan MD, PhD,Steven M. Dubinett MD,Jay Moon Lee MDe 1960s, typically in the fundamental memoir "The Non-Linear Field Theories of Mechanics" by Truesdell and Noll. The theoretical aspect of constitutive theories for materials in general has been carefully developed in mathemati- cal clarity - from general kinematics, balance equations, material obje

狂怒

e 1960s, typically in the fundamental memoir "The Non-Linear Field Theories of Mechanics" by Truesdell and Noll. The theoretical aspect of constitutive theories for materials in general has been carefully developed in mathemati- cal clarity - from general kinematics, balance equations, material obje

MITE

Inflammation and Lung Cancer: The Relationship to Chronic Obstructive Pulmonary Disease,dence even after correcting for cigarette smoke consumption. The mechanistic explanations for this epidemiologic link remain poorly understood as the two disease processes are seemingly polar opposites. However, there are key shared pathologic mechanisms that may link COPD and lung cancer, and infla

大范圍流行

,Inflammation and Lung Cancer: The Role of Epithelial–Mesenchymal Transition, Physiologic EMT is an essential part of normal embryonic development and an adult organism’s ability to overcome acute injury. However, chronic injury and inflammation can yield dysregulated or pathologic EMT that drives organ fibrosis and cancer development. In this chapter, we review seminal work

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