Titlebook: Inflammation; John R. Vane,Sergio H. Ferreira Book 1978 Springer-Verlag Berlin Heidelberg 1978 Chemokine.Histamin.Hypothalamus.Insulin.Nic

強烈興趣

書目名稱Inflammation影響因子(影響力)




書目名稱Inflammation影響因子(影響力)學科排名




書目名稱Inflammation網(wǎng)絡公開度




書目名稱Inflammation網(wǎng)絡公開度學科排名




書目名稱Inflammation被引頻次




書目名稱Inflammation被引頻次學科排名




書目名稱Inflammation年度引用




書目名稱Inflammation年度引用學科排名




書目名稱Inflammation讀者反饋




書目名稱Inflammation讀者反饋學科排名

膽汁

Complemente. Detailed studies of the human complement sequence have facilitated the demonstration of acquired abnormalities in association with disease, while an understanding of the system in certain animals has provided models for experimental studies of the role of complement in inflammation.

滑動

Bradykinin-Systemradykinin (Bk), in such reactions will be discussed. The main scope is to evaluate to what extent kinins are contributing to the inflammatory response, as well as to assess the significance of their participation in such a complex, multimediated process.

出生

STALL

The Sequence of Early Eventsd by all other techniques—histological, ultrastructural, biochemical or pharmacological—must be compared in order to determine its relevance to the morphology and mechanisms of the tissue response to injury.

DOLT

Immunological and Para-Immunological Aspects of Inflammationconcerned with a review of the immunological causes of inflammation and, in addition, with those phenomena which may lack the specificity normally associated with the “specific adaptive immune response” but which use some of the pathways normally associated with immunological reactions, such as the alternative pathway of complement activation.

一瞥

A Brief History of Inflammatione phenomenon was recognized by Galen (3rd century, A.D.) as a reaction of the body against injury, as well as by . (1794), an English doctor of the 18th century. An interesting wealth of historical information about that period can be found in the series of papers by . (1970–1972).

fibroblast

大看臺

高度

The Release of Hydrolytic Enzymes From Phagocytic and Other Cells Participating in Acute and Chronicner towards an extracellular stimulus. Also viable cells under certain conditions, can be induced to release newly synthesized hydrolases over a long period of time, illustrating a possible mechanism by which tissue damage, degradation and remodelling seen during chronic inflammatory processes can occur.