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Titlebook: Immunosuppression and Human Malignancy; David Naor,Benjamin Y. Klein,Jonathan S. Duke-Coha Book 1989 The Humana Press Inc. 1989 Antigen.T

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發(fā)表于 2025-3-21 17:16:59 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Immunosuppression and Human Malignancy
編輯David Naor,Benjamin Y. Klein,Jonathan S. Duke-Coha
視頻videohttp://file.papertrans.cn/463/462290/462290.mp4
叢書名稱Contemporary Immunology
圖書封面Titlebook: Immunosuppression and Human Malignancy;  David Naor,Benjamin Y. Klein,Jonathan S. Duke-Coha Book 1989 The Humana Press Inc. 1989 Antigen.T
描述The immune system can deal effectively with the majority of viruses and bacteria, less effectively with parasites, and very poorly with cancer. Why is this so? Why are McFarlane Burnet‘s and Lewis Thomas‘ predictions that the immune system is in- volved in ridding the body of cancer cells, encapsulated in the catchy phrase "immunologic surveillance," so difficult to experi- mentally establish? Cancer differs from infectious agents in being derived from the host. Hence, it has been postulated that cancer cells lack anti- gens that the immune system can recognize. They are not "im- munogenic. " However, this argument is seriously weakened by the existence of numerous human autoimmune diseases, in which the immune system effectively recognizes and attacks a va- riety of self tissues. Thus, the potential clearly exists for recogni- tion of the surfaces of tumor cells. Professor Naor and his colleagues have written a book that explores another possible reason: cancer cells are recognized by the immune system-but is it possible that the consequence of recognition is inhibition of the immune system-by suppressor T cells or macrophages? The evolution of the malignant state may only occur i
出版日期Book 1989
關(guān)鍵詞Antigen; T cell; autoimmunity; carcinogenesis; cell; diseases; immunosuppression; immunotherapy; leukemia; ly
版次1
doihttps://doi.org/10.1007/978-1-4612-4496-7
isbn_softcover978-1-4612-8846-6
isbn_ebook978-1-4612-4496-7
copyrightThe Humana Press Inc. 1989
The information of publication is updating

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發(fā)表于 2025-3-21 20:15:44 | 只看該作者
Suppressor Cells and Malignancy in Animal Experimental Models,Since our previous review article was published (.), new information concerning suppressor cells in tumor-bearing animals has come our way, most of it published during the years 1985 to 1987. These papers may shed more light on some aspects described in this book and, hence, will be briefly reviewed.
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978-1-4612-8846-6The Humana Press Inc. 1989
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發(fā)表于 2025-3-22 10:19:24 | 只看該作者
Introduction,r-bearing hosts (TBH). It is difficult, however, to evaluate the genuine extent of immunodepression in experimental models of malignancy, and its relationship to concurrent suppressor cell function, simply because negative results are not always reported.
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發(fā)表于 2025-3-22 14:30:17 | 只看該作者
er. Why is this so? Why are McFarlane Burnet‘s and Lewis Thomas‘ predictions that the immune system is in- volved in ridding the body of cancer cells, encapsulated in the catchy phrase "immunologic surveillance," so difficult to experi- mentally establish? Cancer differs from infectious agents in be
7#
發(fā)表于 2025-3-22 19:13:33 | 只看該作者
Induction of Suppressor Cells by Immunostimulants,ling capacity may be found in these cells. Humoral and cellular immune responses may be enhanced in animals treated with BCG (.) or . (.) because of the ability of these immunostimulants to augment the capacity of macrophages to process and present antigen to lymphocytes. In addition, . and BCG can augment the activity of NK cells (.).
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