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Titlebook: Immunopharmacology; Manzoor M Khan Book 2016Latest edition Springer International Publishing Switzerland 2016 auto-immune disease.cytokine

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發(fā)表于 2025-3-21 16:58:56 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Immunopharmacology
編輯Manzoor M Khan
視頻videohttp://file.papertrans.cn/463/462270/462270.mp4
概述Second edition delves into new discoveries in immunopharmacology.Provides a deeper understanding of the recent advances and discoveries of the function on the immune response and their applications in
圖書(shū)封面Titlebook: Immunopharmacology;  Manzoor M Khan Book 2016Latest edition Springer International Publishing Switzerland 2016 auto-immune disease.cytokine
描述.The concept of immunotherapy was in infancy when the first edition was written; since then, major advances have been made, not only with several prominent clinical trials, but also with the approval of cell-based therapy by the FDA for the treatment of cancer in 2010.? These events resulted in a gradually narrowing gap between early scientific knowledge and the late development of immune-based therapies. Consequently, the significance and magnitude of these advances warranted a revision of this contribution; this revised edition will provide a deeper understanding of the recent advances and discoveries related to the function of the immune response and their applications in the development of novel therapies to treat human diseases. Some of the key discoveries during the past five years include: the identification of the new subsets of helper T cells; new cytokines and their networks; and novel signal transduction mechanisms. For example, the identification of TH17 subset of helper T cells, in addition to TH1 and TH2 cells, not only advanced our understanding of the function of the basic immune response, but also raised our awareness of the possible etiology and pathogenesis of di
出版日期Book 2016Latest edition
關(guān)鍵詞auto-immune disease; cytokine networks; helper T cells; immune response; pathogenesis
版次2
doihttps://doi.org/10.1007/978-3-319-30273-7
isbn_softcover978-3-319-80768-3
isbn_ebook978-3-319-30273-7
copyrightSpringer International Publishing Switzerland 2016
The information of publication is updating

書(shū)目名稱Immunopharmacology影響因子(影響力)




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Cytokine Receptors and Signaling,and to its receptor, there is an initiation of the signal transduction pathways culminating in the synthesis of new mRNA and protein synthesis, in most cases. Some of these pathways are unique targets for therapeutic manipulations in disease state. This chapter focuses on five families of cytokine r
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Monoclonal Antibodies as Therapeutic Agents,t and thus is useful to treat a variety of disease states. This chapter begins with an explanation of methods which are used to propagate and isolate monoclonal antibodies. There is description of hybridoma technology, CDR grafting, and phage display technology. These techniques are used to develop
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Allergic Disease,fter exposure to low levels of environmental or food allergens instead of IgG. The first part of this chapter is devoted to defining hypersensitivity disease, IgE-mediated responses, and regulation of IgE synthesis. IgE synthesis is regulated by heredity, natural history of antigen exposure, nature
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Tissue Transplantation, where the transplanted graft is from the same individual or it could be allogeneic, where the graft is transplanted in a genetically different individual. In this chapter, the focus is predominantly on allogeneic transplantation. The ability to distinguish self from nonself resides in the HLA molec
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Acquired Immune Deficiency Syndrome,ncy, which leads to opportunistic infections that are the major cause of death. In this chapter, a brief history of the HIV infection is provided followed by the modes of HIV transmission, the criteria for diagnosis, a description of the components of the virus, and its cell cycle. The life cycle po
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Regulatory T Cells and Disease State, to self-antigens. This chapter describes the nomenclature and function of various types of Treg cells that possess immunosuppressive function. Their major subsets include natural Treg cells, peripheral Treg cells, Tr1 cells, TH3 cells, CD8 Treg cells, Qa-1-restricted CD8., CD8. CD28., and NKT cells
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