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Titlebook: Immunology and Alzheimer’s Diseasee; Colloque Medecine et Annick Pouplard-Barthelaix,Jean Emile,Yves Christe Conference proceedings 1988 Sp

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發(fā)表于 2025-3-21 16:30:03 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Immunology and Alzheimer’s Diseasee
副標(biāo)題Colloque Medecine et
編輯Annick Pouplard-Barthelaix,Jean Emile,Yves Christe
視頻videohttp://file.papertrans.cn/463/462210/462210.mp4
叢書(shū)名稱Research and Perspectives in Alzheimer‘s Disease
圖書(shū)封面Titlebook: Immunology and Alzheimer’s Diseasee; Colloque Medecine et Annick Pouplard-Barthelaix,Jean Emile,Yves Christe Conference proceedings 1988 Sp
描述Alzheimer‘s disease is one of the major scientific, medical and social challenges of our time. This book, the first in the new series .Research and Perspectives in Alzheimer‘s . .Disease., presents a particularly up-to-date approach of immunological and biochemical aspects. It is written by the most outstanding scientists and contains recent data. The following are among the most interesting ideas contained in this book: - Alzheimer‘s disease is a cerebral form of amyloidosis. - some data are in accordance with an immunological hypothesis of this disease. - degeneration of microvessels - including amyloid angiopathy - could be very important in the changes in the brain with Alzheimer‘s disease. - the molecular study of A4 protein and its precursor is important for the understanding of the disease. This synthesis by prominent scientists provides a stimulating hypothesis about the determinism of the disease.
出版日期Conference proceedings 1988
關(guān)鍵詞Alzheimer; alzheimer‘s disease; brain; diseases; genetics; immunoglobulin; immunotherapy; proteins; virus
版次1
doihttps://doi.org/10.1007/978-3-642-46634-2
isbn_softcover978-3-642-46636-6
isbn_ebook978-3-642-46634-2Series ISSN 0945-6066 Series E-ISSN 2196-310X
issn_series 0945-6066
copyrightSpringer-Verlag Berlin Heidelberg 1988
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 23:22:58 | 只看該作者
,Alzheimer’s Disease, a Cerebral Form of Amyloidosis,rch for ways of inhibiting and/or activating the cells producing and/or processing aging-Alzheimer-amyloidogenic protein (AAAP) as well as methods which may help to remove the deposits of amyloid fibers.
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發(fā)表于 2025-3-22 01:09:48 | 只看該作者
,The Molecular Basis of Cerebral Amyloidosis in Alzheimer’s Disease and the Unconventional Virus Dise, which in turn is derived from a neuronal membrane glycoprotein. An understanding of the process by which the PrP molecule is converted into an amyloidogenic molecule may shed some light on the nature of the infectious unit.
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,The Blood-Brain Barrier in Cerebral Amyloidogenesis in Alzheimer’s Disease and Scrapie, on localisation of proteins and antigens. It is concluded that the primary pathogenesis of plaques occurs within the neuroparenchyma and that the evidence for systemic involvement remains inconclusive.
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發(fā)表于 2025-3-23 04:52:28 | 只看該作者
,Immunological Markers and Neuropathological Lesions in Alzheimer’s Disease,ssical pathway (C1, C3, C4) were demonstrated in the brain amyloid lesions of four patients who had Alzheimer’s disease..Staining intensity appeared to depend on the kind of senile plaques. There was a predominance of C1q staining on plaques with a central amyloid core and on amyloid masses around t
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