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Titlebook: Immunological Mechanisms and Therapies in Brain Injuries and Stroke; Jun Chen,Xiaoming Hu,John H. Zhang Book 2014 Springer Science+Busines

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發(fā)表于 2025-3-21 18:54:25 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Immunological Mechanisms and Therapies in Brain Injuries and Stroke
編輯Jun Chen,Xiaoming Hu,John H. Zhang
視頻videohttp://file.papertrans.cn/463/462191/462191.mp4
概述Explores the role of inflammation and immunological mechanisms in protecting the CNS during injury.Looks at translational and clinical research.Provides a comprehensive up-to-date overview.Includes su
叢書名稱Springer Series in Translational Stroke Research
圖書封面Titlebook: Immunological Mechanisms and Therapies in Brain Injuries and Stroke;  Jun Chen,Xiaoming Hu,John H. Zhang Book 2014 Springer Science+Busines
描述Recent research has revealed the importance of immunological mechanisms and inflammation in delaying damage and/or promoting repair after an acute injury to the central nervous system.? This book provides a comprehensive and up-to-date overview of the role of immunological mechanisms and therapies for treating acute neurological injuries such as cerebral ischemia, hemorrhage, and brain and spinal cord trauma. In several sections, the contributing authors provide a review of immunological mechanisms involved in neurological injury and of various translational and clinical research aimed at harnessing those mechanisms for better patient outcomes.?
出版日期Book 2014
關(guān)鍵詞adhesion molecules; biomarkers; chemokines; cytokines; immunological mechanisms
版次1
doihttps://doi.org/10.1007/978-1-4614-8915-3
isbn_softcover978-1-4939-4495-8
isbn_ebook978-1-4614-8915-3Series ISSN 2363-958X Series E-ISSN 2363-9598
issn_series 2363-958X
copyrightSpringer Science+Business Media New York 2014
The information of publication is updating

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978-1-4939-4495-8Springer Science+Business Media New York 2014
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Immunological Mechanisms and Therapies in Brain Injuries and Stroke978-1-4614-8915-3Series ISSN 2363-958X Series E-ISSN 2363-9598
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發(fā)表于 2025-3-22 12:45:43 | 只看該作者
https://doi.org/10.1007/978-1-4614-8915-3adhesion molecules; biomarkers; chemokines; cytokines; immunological mechanisms
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Immune Cell-Derived Free Radicals in Acute Brain Injury,emia. Brain injuries like ischemic–reperfusion result in a surge of excess oxygen that leads to generation of free radicals. Free radicals are present at low levels in the normal state where they play a critical role in signaling pathways. Antioxidants help in maintaining the redox level in the cell
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