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Titlebook: Immune Deficiency and Cancer; Epstein-Barr Virus a David T. Purtilo Book 1984 Plenum Publishing Corporation 1984 antigen.cancer.cancer regi

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發(fā)表于 2025-3-21 18:58:08 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Immune Deficiency and Cancer
副標題Epstein-Barr Virus a
編輯David T. Purtilo
視頻videohttp://file.papertrans.cn/463/462045/462045.mp4
圖書封面Titlebook: Immune Deficiency and Cancer; Epstein-Barr Virus a David T. Purtilo Book 1984 Plenum Publishing Corporation 1984 antigen.cancer.cancer regi
描述The discoveries of Burkitt, Epstein, and Henle have laid the foundation for continuing generation of information regarding the mechanisms of induction of diseases by Epstein-Barr virus. The discovery of the virus two decades ago resulted from clinical and basic science collaborative studies on Burkitt lymphoma. Subse- quently, nasopharyngeal carcinoma and infectious mononucleosis have been linked etiologically with the virus. During the first decade of research following the discovery of the virus, the mechanisms for the induction of BL, NPC, and IM were sought. At that time one prevailing view was that individual oncogenic strains of EBV were responsible for the different disorders. Paralleling the development of immunology in the 1970‘s was the accrual of knowledge about immunological events occurring during IM. These studies suggest that immune defense mechanisms deter- mine the outcome of this viral infection rather than different viral strains. During the early 1970‘s, Starzl and Penn and Gatti and Good had noted an increased frequency of malignancy in renal allograft recipients and children with primary immune deficiency disorders, respectively. These observations provoked in
出版日期Book 1984
關(guān)鍵詞antigen; cancer; cancer registry; carcinoma; cell; children; diseases; immunology; infectious; interferon; lym
版次1
doihttps://doi.org/10.1007/978-1-4684-4760-6
isbn_softcover978-1-4684-4762-0
isbn_ebook978-1-4684-4760-6
copyrightPlenum Publishing Corporation 1984
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 21:10:55 | 只看該作者
板凳
發(fā)表于 2025-3-22 03:28:49 | 只看該作者
Immune Deficiency, Epstein-Barr Virus (EBV) and Lymphoproliferative Disordersated with EBV (1). Empirical evidence that immune deficiency is important in lymphomagenesis stems from reports of lymphomas in renal transplant recipients and in children with inherited immunodeficiency (2).
地板
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Hematopathology of X-Linked Lymphoproliferative Syndromelogical manifestations of the major phenotypes of XLP; and 3) briefly consider other potential oncogenic viruses which may provoke opportunistic malignancies in individuals with acquired or inherited immunodeficiency.
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發(fā)表于 2025-3-23 00:27:23 | 只看該作者
Lymphoma in Cardiac Transplant Recipients Associated with Cyclosporin A, Prednisone and Anti-Thymocyct T cell suppressoV responses, was substituted for azathioprine in the conventional therapy regimen (cyclosporin A therapy) (1,2). Although outcome of transplantation has been favorably effected in patients treated with cyclosporin A (79% one year survival vs. 63% in conventionally treated recipients) morbidity due to lymphoma has increased.
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發(fā)表于 2025-3-23 03:11:20 | 只看該作者
Immunodeficiency to Epstein-Barr Virus in Chediak-Higashi Syndromephilic granules in polymorphonuclear neutrophils and lymphocytes were seen. Based on the symptoms, “Downey-McKinley” cells, and a positive Paul Bunnell reaction in one case, Cesar postulated that infectious mononucleosis (IM) “an organism in the patient lacking host defenses, was the reason...for the bone marrow failure.”
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發(fā)表于 2025-3-23 05:42:02 | 只看該作者
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