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Titlebook: IFS; Conditionals, Belief William L. Harper,Robert Stalnaker,Glenn Pearce Book 1981 D. Reidel Publishing Company, Dordrecht, Holland 1981 J

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發(fā)表于 2025-3-21 16:53:15 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱IFS
副標(biāo)題Conditionals, Belief
編輯William L. Harper,Robert Stalnaker,Glenn Pearce
視頻videohttp://file.papertrans.cn/461/460267/460267.mp4
叢書名稱The Western Ontario Series in Philosophy of Science
圖書封面Titlebook: IFS; Conditionals, Belief William L. Harper,Robert Stalnaker,Glenn Pearce Book 1981 D. Reidel Publishing Company, Dordrecht, Holland 1981 J
描述With publication of the present volume, The University of Western Ontario Series in Philosophy of Science enters its second phase. The first fourteen volumes in the Series were produced under the managing editorship of Professor James J. Leach, with the cooperation of a local editorial board. Many of these volumes resulted from colloguia and workshops held in con- nection with the University of Western Ontario Graduate Programme in Philosophy of Science. Throughout its seven year history, the Series has been devoted to publication of high quality work in philosophy of science con- sidered in its widest extent, including work in philosophy of the special sciences and history of the conceptual development of science. In future, this general editorial emphasis will be maintained, and hopefully, broadened to include important works by scholars working outside the local context. Appointment of a new managing editor, together with an expanded editorial board, brings with it the hope of an enlarged international presence for the Series. Serving the publication needs of those working in the various subfields within philosophy of science is a many-faceted operation. Thus in future the Serie
出版日期Book 1981
關(guān)鍵詞Jackson Pollock; comparative; conditional; probability; subject; time
版次1
doihttps://doi.org/10.1007/978-94-009-9117-0
isbn_softcover978-90-277-1220-2
isbn_ebook978-94-009-9117-0Series ISSN 1566-659X Series E-ISSN 2215-1974
issn_series 1566-659X
copyrightD. Reidel Publishing Company, Dordrecht, Holland 1981
The information of publication is updating

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hydroxymethylcytosine, formylcytosine, carboxylcytosine) levels, and histone modifications. This chapter examines epigenetic alterations beyond the island, and summarizes recent findings in DNA-based epigenetic regulation of the two most commonly diagnosed cancers in the Western world: colorectal ca
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Robert C. Stalnakerhydroxymethylcytosine, formylcytosine, carboxylcytosine) levels, and histone modifications. This chapter examines epigenetic alterations beyond the island, and summarizes recent findings in DNA-based epigenetic regulation of the two most commonly diagnosed cancers in the Western world: colorectal ca
地板
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David Lewishydroxymethylcytosine, formylcytosine, carboxylcytosine) levels, and histone modifications. This chapter examines epigenetic alterations beyond the island, and summarizes recent findings in DNA-based epigenetic regulation of the two most commonly diagnosed cancers in the Western world: colorectal ca
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Robert C. Stalnakeron age or the extent of acceleration with mortality and various aging-related conditions, even?after accounting for differences in chronological age and other risk factors. Although epigenetic profiles are known to be tissue-specific, both target tissue- and multiple tissue-derived estimates appear
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response escape, such as classical HLA proteins and related APM (antigen presentation machinery) with their epigenetic changes contribute to the tumor development process, either to tumor progression or regression, depending on the immune effector cells that are in play.
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發(fā)表于 2025-3-22 20:09:07 | 只看該作者
Robert C. Stalnakern DNA methylation patterns that play a critical role in cancer development. Specific epigenetic changes in esophageal, hepatic, and colorectal cancers have been detected in blood samples and proposed to be used clinically as epigenetic biomarkers for diagnosis and prognosis of these cancers. Also, g
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of oncogenes (e.g., NFAT5, MALAT1, MET, FOXA1, KRAS, S100P, OSTF1) and glutamate transporter gene SLC1A1. TNFAIP8 knockdown cells also exhibited decreased expression of multiple onco-proteins (e.g., PIK3CA, SRC, EGFR, IL5, ABL1, GAP43), and increased expression of the orphan nuclear receptor NR4A1
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