Titlebook: Hypoxia; From Genes to the Be Robert C. Roach,Peter D. Wagner,Peter H. Hackett Book 2001 The Editor(s) (if applicable) and The Author(s), u

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The heme oxygenase system and cellular defense mechanismse molecule, including that of NO synthase. Unknown until recent years was that the system is a central component of the cellular defense mechanisms; this can be attributed to a combination of many factors. In biological systems HO activity is responsible for production of equimolar amounts of CO, bi

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Hypoxia-inducible factor in brainscription of hypoxia-inducible target genes. Because HIF-1 activation might promote cell survival in hypoxic tissues, we studied the effect of stroke on the expression of HIF-1α, HIF-1? and several HIF-1 target genes in adult rat brain. After focal cerebral ischemia, mRNAs encoding HIF-1α, glucose t

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Proton-gated cation channels — neuronal acid sensors in the central and peripheral nervous system inflammation is painful and sensory neurons respond to acidic tissue pH with increased firing. H+-gated cation channels in sensory nerve endings are thought to be responsible for the activation of nociceptive afferents by acid. The members of one family of recently identified H+-gated cation channe

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https://doi.org/10.1057/9780230513310s 12. International Symposium, John T. (Jack) Reeves, MD, was selected to receive this honor. No one is more deserving, and I say this having had the privilege of Jack’s friendship for nearly half a century.

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