Titlebook: Hypertension: Mechanisms and Management; Thomas Philipp,Armin Distler Conference proceedings 1980 Springer-Verlag Berlin Heidelberg 1980 A

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書目名稱Hypertension: Mechanisms and Management影響因子(影響力)




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Plasma Catecholamine Levels and the Renin-Angiotensin-System in Subjects with Borderline Hypertensio noradrenaline is released from nerve endings. The majority of it is taken up again into the neurons but a small fraction reaches the systemic circulation. Thus, plasma noradrenaline concentration can be used as an index of sympathetic nervous activity. In man, most if not all adrenaline in plasma s

angina-pectoris

Cardiovascular Pressor Reactivity as Related to Plasma Catecholamines: Role in the Pathogenesis of E was evaluated by combined analysis of plasma catecholamine levels and pressor sensitivity to norepinephrine (NE)..Under placebo conditions, plasma NE, epinephrine and dopamine concentrations did not differ significantly between normal subjects and patients with borderline or established essential h

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Plasma Noradrenaline as an Index of Sympathetic Activityaken up into neuronal or extraneuronal tissue or metabolised by methylation or deamination and which overflows into the circulation. Plasma adrenaline in contrast is derived from the adrenal medulla and exerts its action as a blood borne hormone on metabolic, endocrine and vascular adrenergic recept

概觀

The Central Peptidergic Stimulation Syndrome: A Role for Brain Peptides in Central Mechanisms of Bloion of ANG II receptors by exogenous administration of the peptide or endogenous stimulation of its biosynthesis leads to the release of antidiuretic hormone (ADH) and adrenocorticotropic hormone (ACTH) and to an increase of sympathetic tone (“central peptidergic stimulation syndrome”). ENK produce

DRAFT

Glandular Adenylate Cyclase System in Genetic Hypertension — Age-Dependent Response to Catecholaminef high arterial blood pressure in spontaneously hypertensive rats (SHR). Both sympatho-neural and sympatho-adrenal malfunctions are involved in causing and possibly maintaining arterial hypertension in SHR and yet, the experimental evidence that has been published to date is often divergent and the

巫婆

Acute Effects of Labetalol on Haemodynamics and Response to Emotional Stress in Hypertensive Patienty the prevalence of a reflex alpha-stimulation. Eight years ago, an agent was introduced under the name of labetalol of which it is claimed that it blocks both the alpha- and beta-receptors [3, 22]. Its beta-blocking properties are thought to be more effective than the alpha-blocking ones [23]. Pero