Titlebook: Hyperfine Interactions (C); Proceedings of the I Michael F. Thomas,John M. Williams,Terence C. Gibb Conference proceedings 2002 Springer Sc

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A M Gismelseed,A A Yousif,A D Al-Rawas,M E Elzain,I Ayub,H. M. Widatallah,F. J. Berryto the molecular phenotype of an individual tumor, with each patient receiving a different combination of therapeutics aimed at the specific aberrant proteins driving the cancer. Tracking the a978-1-4899-8451-7978-1-4419-5714-6

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H.-C. Wille,M. Gerken,E. Gerdau,Yu. V. Shvyd’ko,H. D. Rüter,H. Franzpecific proteins in each pathway mutated or differentially expressed in a given tumor (The Cancer Genome Atlas Research Network 2008; Parsons et al. 2008). Different studies demonstrated that while widespread mutations exist in cancer, not all mutations drive cancer development (Lin et al. 2007). Th

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Y. Yoda,S. Kishimoto,X. W. Zhang,M. Seto,S. Kikuta differentially expressed in a given tumor (The Cancer Genome Atlas Research Network 2008; Parsons et al. 2008). Different studies demonstrated that while widespread mutations exist in cancer, not all mutations drive cancer development (Lin et al. 2007). This suggests a need to target only a deleter

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T. Asthalter,I. Sergueev,H. Franz,W. Petry,K. Messel,R. Verbenipecific proteins in each pathway mutated or differentially expressed in a given tumor (The Cancer Genome Atlas Research Network 2008; Parsons et al. 2008). Different studies demonstrated that while widespread mutations exist in cancer, not all mutations drive cancer development (Lin et al. 2007). Th