Titlebook: Human Cell Transformation; Advances in Cell Mod Johng S. Rhim,Anatoly Dritschilo,Richard Kremer Book 2019 The Editor(s) (if applicable) and

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Novel Human Prostate Epithelial Cell Cultureser incidence and mortality rate from prostate cancer compared to Caucasian men in North America, indicating the prostate cancer is a major public health problem in this population. Studies of prostate cancer have been hampered by various factors including (1) restricted access to tissues, (2) diffic

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Assessing the Advantages, Limitations and Potential of Human Primary Prostate Epithelial Cells as a nale that can be behind this decision and we summarize the current cell models that are used to study prostate cancer. Researchers face the challenge of choosing a model that recapitulates the complexity and heterogeneity of prostate cancer. The use of primary prostate epithelial cells cultured from

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Role of Alternative Splicing in Prostate Cancer Aggressiveness and Drug Resistance in African Americies diversity. Dysregulation of alternative splicing can initiate and drive disease. Aberrant alternative splicing has been shown to promote the “hallmarks of cancer” in both hematological and solid cancers. Of interest, recent work has focused on the role of alternative splicing in prostate cancer

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Discovery of Metabolic Biomarkers Predicting Radiation Therapy Late Effects in Prostate Cancer Patie. Management options may include watchful waiting, surgery, or radiation therapy. Thus, initial risk stratification of prostate cancer patients is important for achieving optimal therapeutic results or cancer cure and preservation of quality of life. Predictive biomarkers for risks of complications

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The Aging Skeletonetal sites and varies between sexes. Accelerated loss of bone occurs in the perimenopausal period in women, whereas more gradual but progressive loss of bone occurs in aging men. Changes in bone quality as well as bone quantity occur during growth and subsequent aging. These include changes in bone

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Targeting DNA Hypomethylation in Malignancy by Epigenetic Therapiesin various cellular processes like embryogenesis, genomic imprinting, X-chromosome inactivation, and genome stability. Alterations in the normal methylation levels of DNA may contribute to the development of pathological conditions like cancer. Even though both hypo- and hypermethylation-mediated ab

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