Titlebook: Hughes Syndrome; Antiphospholipid Syn M. A. Khamashta (Deputy Head) Textbook 20001st edition Springer-Verlag London 2000 Antiphospholipid S

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R. G. Lahitateen short-term bioassays. The genetic endpoints used included point or gene mutation, primary DNA damage, and chromosomal effects. Initially, all pesticides were tested in a “core” battery of four . bioassays. A carefully selected group among these chemicals was retested in higher level test system

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Antiphospholipid Syndrome: General Featuresristic clinical features of this syndrome although the spectrum of associated symptoms and signs has broadened considerably over the last 15 years. The aim of this chapter is to give a clinical overview and preliminary classification criteria for the APS by way of introduction to the more detailed c

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The Eye in the Primary Antiphospholipid Syndrome (Hughes Syndrome)ocytopenia and multi-organ compromise [1–13]. Vessel occlusion is a hallmark of this association, it may occur within the context of several diseases, mainly autoimmune disorders such as systemic lupus erythematosus, or it may be present without any recognizable disease, the so called primary antiph

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The Primary Antiphospholipid Syndromeunity. The identification of a pure or “primary” variant of the syndrome has been central to this story, not least in enabling us to establish the place of this remarkable condition in the wide spectrum of autoimmune disease.