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Titlebook: Host Defenses and Immunomodulation to Intracellular Pathogens; Toby K. Eisenstein (Professor of Microbiology and Book 1988 Springer Scien

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書目名稱Host Defenses and Immunomodulation to Intracellular Pathogens
編輯Toby K. Eisenstein (Professor of Microbiology and
視頻videohttp://file.papertrans.cn/429/428402/428402.mp4
叢書名稱Advances in Experimental Medicine and Biology
圖書封面Titlebook: Host Defenses and Immunomodulation to Intracellular Pathogens;  Toby K. Eisenstein (Professor of Microbiology and  Book 1988 Springer Scien
出版日期Book 1988
關(guān)鍵詞Host; host defenses; immunomodulation; intracellular pathogens
版次1
doihttps://doi.org/10.1007/978-1-4757-5421-6
isbn_softcover978-1-4757-5423-0
isbn_ebook978-1-4757-5421-6Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightSpringer Science+Business Media New York 1988
The information of publication is updating

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0065-2598 Overview: 978-1-4757-5423-0978-1-4757-5421-6Series ISSN 0065-2598 Series E-ISSN 2214-8019
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Advances in Experimental Medicine and Biologyhttp://image.papertrans.cn/h/image/428402.jpg
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and , Role of Gamma Interferon in Immune Clearance of ,Members of the genus . are obligate intracellular bacteria, and due to the intracellular location of their growth, they present a unique challenge to the immune system. Since antibody cannot gain entrance to viable cells, it has been argued that antibody does not play a major role in the effector phase of immunity to these organisms.
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Genetic Basis of Host Resistance and Susceptibility to Intracellular Pathogens and in the endemic areas of the world. Although the influence of environmental factors must be considered in any explanation of such variability, it has recently become clear, mainly on the basis of studying experimental infections in inbred animals, that genetic factors play decisive role in individual susceptibility.
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Cutaneous Leishmaniasis: Relative Role of T Cell Subsetsectrum of clinical manifestations can be observed in patients suffering from each of these forms of the disease which suggests that the pathologic changes observed in leishmaniasis are due to both the species of the infecting parasite and to the host response to the parasite.
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