Titlebook: Hormones and Cancer; Wendell W. Leavitt Book 1982 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Sci

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A. M. H. Brodie,H. J. Brodie,L. Romanoff,J. G. Williams,K. I. H. Williams,J. T. Wu

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Etiology of DES-Induced Uterine Tumors in the Syrian Hamster,ss of the DES-altered uterus to estrogen and progestin action was not impaired. Morphogenetic changes included an increase in extracellular connective tissue elements and striking alterations in endometrial cell composition such as hyperplasia of luminal and glandular epithelia and a massive inflamm

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Biologic Activity of the Iodoestrogens and Their Use in Breast Cancer,stration of (.) to rats with DMBA-induced mammary tumors, no tumor concentration of radioactivity could be detected by imaging. When (.) was administered similarly, radioactivity could be detected in some of the tumors by imaging. The radioactivity was associated with non-specific 4S proteins.

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Retinoid Binding in Normal and Neoplastic Mammary Tissue,nditions, translocates into the nucleus. Unbound [.H]retinoic acid, however, failed to associate with the nuclear sites. Additional studies indicate a possible selective inhibition of ovarian hormone independent tumors by the retinoids.

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Feedback Effects of Mammary Gland Tumors on the Host Pituitary Prolactin Cell,. culture yields data which suggest that the brain of the tumor bearing rat suppresses PRL release. It is suggested that decreased PRL cell function in animals bearing certain mammary tumors ultimately favors metastatic activity of the tumor cell.