Titlebook: Hormonal Carcinogenesis II; Proceedings of the S J. Jonathan Li (Professor of Pharmacology, Toxicol Conference proceedings 1996 Springer-Ve

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Robert W. Haile,Sue Ingles,Victoria K. Cortessis,Robert C. Millikan,Anh Diep,Robert S. Sparkes further readings in the topics of this book to enable the interested reader to delve deeper into the field. Table of Contents: Introduction and overview / Dependence analysis, dependence graphs and alias analysis / Program parallelization / Transformations to modify and eliminate dependences / Tran

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群居動物

Symposium Presentationxplain 1) how hormones regulate mammary epithelial cell proliferation; 2) why hormones are required for the genesis of mammary cancers of heterogeneous phenotypes and genotypes, including those classified as hormone dependent or hormone independent; and 3) why mice, rats, and humans have consistentl

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Symposium Presentatione important regulators of cytochrome P-450; thus, hormonal carcinogenesis may involve hormonal regulation of cytochrome P-450. We have chosen to illustrate regulation of cytochrome P-450 with two examples. The first is the peroxisome proliferator-activated receptor (PPAR), which regulates P-450 of t

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Cell Cycle Controlontrol of the cell cycle has been conserved in all eukaryotic organisms (5). In this chapter, I will attempt to describe these fundamental mechanisms that control the cell cycle, and to relate them to the etiology of cancer development.