Titlebook: Heme Oxygenase in Biology and Medicine; Nader G. Abraham Book 2002 Springer Science+Business Media New York 2002 DNA.genes.genetics.molecu

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Correlation of the Altered Vascular Effects of Carbon Monoxide and the Cardiovascular Complications y as . proposed that CO might possess physiologically important vasoactive functions. That pioneer hypothesis has gained more and more support. The vasorelaxing effects of CO have been observed in many vascular preparations. Several features of the vasorelaxant effects of CO have also been described

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Endogenous Carbon Monoxide has Protective Roles in Neointimal Development Elicited by Arterial Injurd-othelial damage is believed to be one of the main events involved in the initiation of atherosclerosis. Although various types of growth factor and cytokines, including endothelin-1 (ET-1), platelet-derived growth factor-B (PDGF-B) and angiotensin II (Ang II), have been acknowledged to contribute

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End Tidal Breath Carbon Monoxide (ETCO) Levels in Pregnant WomenNO, stimulates soluble guanylyl cyclase and thereby raises intracellular levels of cyclic guanosine monophosphate (cGMP). Endogenous CO production under physiological conditions serves to regulate smooth muscle tone,. and may play a role in neurotransmission.. Exogenously added CO has been shown to

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Increased Carbon Monoxide in Exhaled Air in Patients with Inflammatory Respiratory Diseasesggregation,. and to act as a neural messenger in the brain..,. CO is produced endogenously in many tissues of the body by the class of enzymes known collectively as heme oxygenase (HO).. Two forms of HO have been characterized. Of these, HO-1 is present in the pulmonary vascular endothelium,. alveol

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Differential Expression of Heme Oxygenase-1 in Rat Brain by Endotoxin (LPS)can produce nitric oxide in response to cytokines, and it is known that neurons, which are characterized by low levels of natural antioxidants and free radical scavengers, are more susceptible to oxidative damage. Heme oxygenase-1 (HO-1) is a cellular stress protein (HSP32) which is expressed in sev

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