Titlebook: Helicobacter pylori; Basic Mechanisms to Richard H. Hunt (Professor of Medicine, Director), Book 1994 Springer Science+Business Media Dord

磨坊

Accolade

FER

ureaseic identification systems for the speciation of bacteria in the clinical laboratory. Over the past decade, however, the role for this enzyme in pathogenesis of many bacterial infections has been recognized (for review see ref. 1).

surrogate

Basic bacteriology of ,: , colonization factorsen specific . antimicrobial therapy or the stomach becomes an inhospitable environment due to such events as changes in the stomach due to long-term gastritis or surgery. Most pathogenic bacteria either infect the host transiently until they are eradicated due to the nonspecific and specific immune

larder

卷發(fā)

嚴厲批評

Reinfection with ,microbial therapy (recrudescence of the infection). Cure of the infection does not guarantee that the individual will not be re-exposed to the bacterium and become reinfected with either the same or a new strain of . Discovery of return of infection after a course or courses of antimicrobial therapy

isotope

faucet

Breakdown of the mucus layer by ,tive mechanisms operating within pre-epithelial, epithelial and post-epithelial compartments. Since most aggressive factors operate within the lumen of the gastrointestinal tract, pre-epithelial defence seems to be a vanguard of mucosal protection and the target absorbing the major impetus of aggres

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