Titlebook: Handbook of Oxidative Stress in Cancer: Mechanistic Aspects; Sajal Chakraborti,Bimal K. Ray,Susanta Roychoudhur Reference work 2022 Spring

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Roles of β-Glucans in Oxidative Stress and Cancere cells, and activate pro-oncogenic signaling pathways. On the other hand, high levels of ROS can contribute to cancer cell death, promoting apoptosis and necrosis. β-Glucans, such as the (1?→?3)-β-D-glucans, have been described as effective in treating dyslipidemia, diabetes, cardiovascular-related

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Functional Regulation Between Matrix Metalloproteases and Cell Junction Proteins in Gastric Cancerdin and occluding. Moreover, . infection and other extrinsic factors impair tight junction which lead to the activation of inflammatory cytokines. Further, mislocalization or altered function of gap junction and adherens junction proteins also responsible for gastric inflammation ultimately leads to

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Association of Oxidative Stress and Mitochondrial Dysfunction to Gynecological Malignanciesents, as per reports, exert dual role by exhibiting both tumor suppressing and tumor promoting functions. Low levels of ROS mainly exert cytotoxic effects on tumor cells, whereas fairly increased ROS levels contribute to cancer progression majorly by remodeling the microenvironment niche, rewiring t

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Targeting Oxidative Stress in Cancercells. Modulation of reactive oxygen species (ROS) is highly significant for cancer therapy because commonly used radio- and chemotherapeutic drugs influence tumor outcome through ROS modulation. In addition, natural compounds, synthetic compounds, and cancer stem cell therapy also play a vital role

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Cutaneous Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stressrogramming which adjusts the protein folding machinery of ER according to the need of a cell. However, in case of failure to re-establish ER homeostasis, the UPR system may activate its pro-apoptotic component leading to disease development. Environmental stressors are diverse and trigger ER stress