Titlebook: Handbook of Copper Pharmacology and Toxicology; Edward J. Massaro Book 2002 Springer Science+Business Media New York 2002 Alzheimer.aquati

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Dental Biofilms in Health and Disease state. Free copper, particularly Cu(I), can catalyze the formation of the highly reactive hydroxyl radicals that damage many cell components, including membranes, proteins, and nucleic acids .. The toxic effects of copper are demonstrated in another human genetic disorder, Wilson’s disease (WD). Th

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Cytochrome-, Oxidase, many articles have been published on its structure and function, isolated from various organisms and tissues, in the last 75 yr or so . since its discovery by Warburg .. Progress in understanding of the reaction mechanism of cytochrome-. oxidase has been accelerated by X-ray structures of the enzy

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The Identification of Motifs Involved in the Intracellular Trafficking of the Menkes Disease Proteinr is much reduced, and the brain, blood, and liver show decreased levels of copper. However, in many other tissues, there is an increase in copper levels when compared to normal individuals, although there is still a defect in the function of copper-requiring enzymes .. Cultured fibroblasts from pat

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Molecular Basis of Diseases of Copper Homeostasis state. Free copper, particularly Cu(I), can catalyze the formation of the highly reactive hydroxyl radicals that damage many cell components, including membranes, proteins, and nucleic acids .. The toxic effects of copper are demonstrated in another human genetic disorder, Wilson’s disease (WD). Th