Titlebook: HDL Metabolism and Diseases; Lemin Zheng Book 2022 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Na

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Carcinogenesis

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ABCA1, ABCG1, and Cholesterol Homeostasis,ulation is toxic to cells and constitutes the molecular basis for many diseases, especially atherosclerotic cardiovascular disease. Thus, cellular cholesterol is tightly regulated to maintain a homeostasis. Reverse cholesterol transport (RCT) is thought to be one primary pathway to eliminate excessi

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HDL and Diabetes,our-fold increased risk of developing cardiovascular disease. This chapter focuses on the anti-diabetic and cardioprotective functions of plasma high-density lipoproteins (HDLs). HDLs and the main HDL apolipoprotein, apoA-I, improves pancreatic beta cell function. ApoA-I also improves insulin sensit

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HDL Mimetic Peptides,w to improve HDL function rather than its plasma level has become a new focus of scientists’ attention. Numerous HDL mimetic peptides have been designed and investigated in various animal models in recent years. Although the underlying mechanisms are not fully understood, the peptides’ antiatherogen

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HDL and microRNAs,to prevent several metabolic diseases. Besides the classic genes regulating cholesterol efflux and reverse cholesterol transport (RCT), microRNAs (miRNAs) could also affect HDLs biogenesis, metabolism, and functions. This chapter summarizes the miRNAs, which regulate HDLs functions in table. In addi

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