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Titlebook: HDL Deficiency and Atherosclerosis; G. Assmann Book 1995 Kluwer Academic Publishers 1995 Lipoprotein.atherosclerosis.cardiovascular.corona

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發(fā)表于 2025-3-25 03:52:23 | 只看該作者
22#
發(fā)表于 2025-3-25 09:33:50 | 只看該作者
Connection between cholesterol efflux and atherosclerosis, The two major subclasses include particles that contain both apoA-I and apoA-II (termed LpA-I, A-II) and those that contain apoA-I but not apoA-II (termed LpA-I). Apo-specific particles can be isolated by use of immunoaffinity columns with anti-apoA-I or anti-apoA-II antibody..
23#
發(fā)表于 2025-3-25 14:06:19 | 只看該作者
https://doi.org/10.1057/9781137295866 possesses arginine. The apoE2 isoform, associated with type III hyperlipoproteinaemia., differs from apoE3 only at residue 158, where it possesses cysteine. Apolipoproteins E3 and E4 bind normally to the low density lipoprotein (LDL) receptor, whereas apoE2 does not.
24#
發(fā)表于 2025-3-25 15:50:58 | 只看該作者
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發(fā)表于 2025-3-25 23:18:23 | 只看該作者
Book 1995on of massive scientific research, continuous transfer of the results to medical practice, community actions and population aware- ness. The death rate from coronary heart disease in the United States rose 20% from 1950 to 1963, when a dramatic and steady downward inflection began, arriving today at
26#
發(fā)表于 2025-3-26 01:16:32 | 只看該作者
HDLC as a therapeutic target in coronary disease: current concepts and future directions,support the concepts that HDL-related measures are as important as LDL-related ones in predicting vascular events. Furthermore, pharmacological or genetic modification of certain plasma HDL subfractions appears to hold promise for clinical benefits that are comparable to, or perhaps exceed, those of LDLC reduction.
27#
發(fā)表于 2025-3-26 05:08:55 | 只看該作者
28#
發(fā)表于 2025-3-26 09:51:21 | 只看該作者
HDL Deficiency and Atherosclerosis978-94-011-6585-3Series ISSN 0166-9842
29#
發(fā)表于 2025-3-26 13:04:37 | 只看該作者
30#
發(fā)表于 2025-3-26 17:31:39 | 只看該作者
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