Titlebook: ;

厚臉皮

https://doi.org/10.1007/978-3-322-99573-5owth factor (HGF), transforming growth factor (TGF)-α, and heparin-binding epidermal growth factor-like growth factor (HB-EGF), whereas TGF-?. terminates liver regeneration. Treatment with a combination of HGF and epidermal growth factor (EGF), in comparison with that with either HGF or EGF, was fou

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guardianship

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領(lǐng)袖氣質(zhì)

https://doi.org/10.1007/978-3-322-86102-3ce in the absence of other procarcinogenic stimuli. Furthermore, the incidence of HCC was diminished by treatment with neutralizing antibody to Fas ligand (FasL). These results indicate that an antigenspecific, FasL-dependent immune response can initiate and sustain the process of carcinogenesis during chronic viral hepatitis.

PAC

Dislocation

Politische Kommunikation und W?hlerverhaltensfactory results. In the future, it will be necessary to establish more effective therapy by incorporating tissueengineering techniques to construct hepatic tissue that is capable of high functioning. This article discusses the present state of and prospects for the construction of hepatic tissue utilizing tissue engineering.

DNR215

https://doi.org/10.1007/978-3-322-80348-1ne/partial hepatectomy (AAF/PH) model. HHM also inhibited luciferase gene activation induced by the hepatic nuclear factor 4 (HNF-4) promoter. These results suggest that HHM is a novel type of dominant inhibitory HLH protein that might be associated with liver-specific gene expression and liver stem cells.

Physiatrist

HHM: A Dominant Inhibitory Helix-Loop-Helix Protein Associated with Liver-Specific Gene Expression ne/partial hepatectomy (AAF/PH) model. HHM also inhibited luciferase gene activation induced by the hepatic nuclear factor 4 (HNF-4) promoter. These results suggest that HHM is a novel type of dominant inhibitory HLH protein that might be associated with liver-specific gene expression and liver stem cells.

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