Titlebook: Gonococci and Meningococci; Epidemiology, Geneti J. T. Poolman Book 1988 Kluwer Academic Publishers, Dordrecht 1988 epidemiology.genetics.p

AVID

Epidemiology of meningococcal (sub)types in the Netherlands 3/100,000 pop./year, which is still below the epidemic levels reached in 1946–1947 and in 1966. The serogroup . increased from 98 strains in 1982 to 275 strains in 1986 (an increase of 281%) and its fraction among the isolated strains rose from 47 to 77%. The B: 15:p1.16 complex was not the driving

Peculate

其他

Plasma LPS and clinical pictures in meningococcal diseasecyte lysate (.) and a chromogenic substrate assay. High initial LPS levels (800–170 x 10. pg/ml) were found in 7 of 8 patients with septic shock, adult respiratory distress syndrome (.) and renal dysfunction. Four of these patients died. Initial . levels were low (85–260 pg/ml) in 3 and absent in 5

inclusive

bourgeois

Intercontinental spread of , clones of the ET-5 Complexoding enzymes in 688 isolates. Variation among strains in serogroup and serotype has little relationship to the complex structure of populations revealed by enzyme electrophoresis, which involves 14 major lineages of clones diverging from one another at more than half their genetic loci. Clones of o

有機體

受傷

A clonal analysis of , serogroup Aection consisting of 423 group A strains isolated from 23 epidemics or outbreaks since 1963, as well as from older epidemics and numerous non-epidemic situations was used in the analysis. Strains were first segregated into electrophoretic types, depending on the combined score for the electrophoreti

玩忽職守

Epidemiology of Infections with Pathogenic , in Swedencal disease per 100,000 inhabitants has varied between 0.9–2.8, the highest figures being reported for 1965 and 1975. With regard to age groups the incidence rates were about 10–15 times higher in infants and young children 1 month — 4 years of age and in the teen age group (15–19 years of age) as c

antipsychotic

Immunoglobulin isotype deficiency and response to meningococcal polysaccharides with those of 17 IgA-replete vaccinates. We measured antibody response to the group A and group C polysaccharides, using a radioactive antigen binding assay. Response among IgA-deficient individuals fell into three categories. 9/17 did not respond, 3 responded normally and the remaining 5 responded

Eructation