Titlebook: Glutamine in Clinical Nutrition; Rajkumar Rajendram,Victor R. Preedy,Vinood B. Pate Book 2015 Springer Science+Business Media New York 201

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Plasma Glutamine and Its Levels in Metabolic Stressy deviate from the normal range, which is associated with an unfavorable outcome. The underlying mechanisms are still obscure. This chapter merely deals with the empirically finding of plasma glutamine concentration in humans in health and disease.

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Glutamine Supplementation in Major Surgery and Intensive Careor the appropriate function of many organs and the principal metabolic fuel for different rapidly dividing cells. Moreover, GLN is essential for cell metabolism because it is able to modulate gluconeogenesis, lipogenesis, anabolic processes, to preserve mitocondrial ATP levels and attenuate the acti

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Enteral Nutrition Supplemented with ,-glutamine in Patients with Sepsis. Fifteen patients received EN supplemented with glutamine (30 g; GLN group) for 2 d followed by EN supplemented with calcium caseinate (30 g, CAS group), also over 2 d. The other 15 patients received EN with calcium caseinate (30 g; CAS group) for 2 d followed by EN with glutamine (30 g; GLN group)

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Glutamine Supplementation in Multiple Trauma Patientsalls dramatically. Therefore, glutamine has been re-classified as an essential amino acid under such catabolic conditions. The role of glutamine supplementation in trauma patients has been extensively studies. Despite the numerous clinical trials that investigated the effect of enteral and parentera

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Glutamine Dipeptide and Insulin Sensitivity in Critically Ill Patientsory metabolites. The de novo acquired hyperglycaemia in these patients is mainly due to decreased insulin sensitivity in liver and muscle. It has been suggested that glutamine, a conditionally essential amino acid in critically ill patients, can influence glucose metabolism, and particularly it can

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Glutamine and Ammonia in Hepatic Encephalopathyscussion of its role in relation to the elevated ammonia levels found in the brain in the context of hepatic encephalopathy (HE), a disturbance of the central nervous system due to liver failure [1] characterized by cognitive, psychiatric, and motor deficits. Early symptoms include reversal of sleep

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,Gro?eltern in der Sicht der Enkel,ersion of glutamate to α-ketoglutarate by a glutamate-linked aminotransferases (or by the action of glutamate dehydrogenase); and 2) conversion of glutamine to α-ketoglutaramate (KGM) catalyzed by glutamine-utilizing transaminases (aminotransferases), followed by conversion of KGM to α-ketoglutarate