Titlebook: Glioma Cell Biology; Aleksi Sedo,Rolf Mentlein Book 2014 Springer-Verlag Wien 2014 Brain.Glioblastoma.Glioma.Tumor.Tumor Angiogenesis.Tumo

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https://doi.org/10.1007/978-3-7091-1431-5Brain; Glioblastoma; Glioma; Tumor; Tumor Angiogenesis; Tumorigenesis

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978-3-7091-4841-9Springer-Verlag Wien 2014

indigenous

Cancer Stem Cells and Glioblastoma to achieve “cure” and was based on the idea that the vast majority of cells have tumorigenic potential. One reason for the lack of clinical advances is the lack of understanding of the GBM biology in general and the cellular origin of this disease in particular. The cancer stem cell hypothesis post

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RAGE

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使迷惑

Para- and Autocrine Mediators in the Glioma Microenvironmentprogression. Growth factor receptors on glioma cells are partly overexpressed or constitutively activated through mutations, and therefore appear prominent targets for anti-glioma therapies. Several cytokines are heavily produced by glioma cells and exhibit not only immune-modulatory functions, but

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oxidant

Microglia in Gliomas: Friend or Foe?ir phenotype, and contribution to glioma pathology. Recent attempts to determine a profile of cytokine/chemokine production and gene expression profiling in CD11b. cells isolated from patients or rodent gliomas revealed their similarity to alternatively activated, the M2-type macrophages observed in

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